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独脚金内酯介导的免疫引发增强了对……的抗病性。 （你提供的原文不完整，这里是根据现有内容尽量通顺翻译的，完整准确的翻译需补充完整原文信息）

Enhanced disease resistance against by strigolactone-mediated immune priming in .

作者信息

Fujita Moeka, Tanaka Tomoya, Kusajima Miyuki, Inoshima Kengo, Narita Futo, Nakamura Hidemitsu, Asami Tadao, Maruyama-Nakashita Akiko, Nakashita Hideo

机构信息

Graduate school of Bioscience and Biotechnology, Fukui Prefectural University.

Graduate School of Bioresource and Bioenvironmental Sciences, Kyushu University.

出版信息

J Pestic Sci. 2024 Aug 20;49(3):186-194. doi: 10.1584/jpestics.D24-019.

DOI:10.1584/jpestics.D24-019
PMID:39398504
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11464267/
Abstract

Strigolactones (SLs) are a class of plant hormones that play several roles in plants, such as suppressing shoot branching and promoting arbuscular mycorrhizal symbiosis. The positive regulation of plant disease resistance by SLs has recently been demonstrated by analyses using SL-related mutants. In Arabidopsis, SL-mediated signaling has been reported to modulate salicylic acid-mediated disease resistance, in which the priming of plant immunity plays an important role. In this study, we analyzed the effect of the synthetic SL analogue -GR24 on resistance against necrotrophic pathogen . In -GR24-treated plants, disease resistance against was enhanced in an ethylene- and camalexin-dependent manners. Expression of the ethylene-related genes and the camalexin biosynthetic gene and camalexin accumulation after pathogen infection were enhanced by immune priming in -GR24-treated plants. These suggest that SL-mediated immune priming is effective for many types of resistance mechanisms in plant self-defense systems.

摘要

独脚金内酯（SLs）是一类植物激素，在植物中发挥多种作用，如抑制枝条分枝和促进丛枝菌根共生。最近，通过使用与SL相关的突变体进行分析，证明了SLs对植物抗病性的正向调节作用。在拟南芥中，据报道SL介导的信号传导可调节水杨酸介导的抗病性，其中植物免疫的启动起着重要作用。在本研究中，我们分析了合成的SL类似物-GR24对抵抗坏死性病原体的影响。在经-GR24处理的植物中，对的抗病性以乙烯和camalexin依赖性方式增强。经-GR24处理的植物在病原体感染后，乙烯相关基因和camalexin生物合成基因的表达以及camalexin的积累通过免疫启动而增强。这些表明SL介导的免疫启动对植物自我防御系统中的多种抗性机制有效。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/c04427d9cb12/jps-49-3-D24-019-figure05.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/dc7460a4f80c/jps-49-3-D24-019-figure01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/53a0c5eeb4cd/jps-49-3-D24-019-figure02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/89c27d22a035/jps-49-3-D24-019-figure03.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/469410e595c1/jps-49-3-D24-019-figure04.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/c04427d9cb12/jps-49-3-D24-019-figure05.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/dc7460a4f80c/jps-49-3-D24-019-figure01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/53a0c5eeb4cd/jps-49-3-D24-019-figure02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/89c27d22a035/jps-49-3-D24-019-figure03.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/469410e595c1/jps-49-3-D24-019-figure04.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6951/11464267/c04427d9cb12/jps-49-3-D24-019-figure05.jpg

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