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TRIM55: An Enemy at the Post-MI Border?: TRIMming May Not Always Be Good.

作者信息

Andreis Marco, Paolocci Nazareno

机构信息

Dipartimento di Scienze Cardio-Toraco-Vascolari e Sanità pubblica, University of Padova, Padova, Italy.

Dipartimento di Scienze Biomediche, Universita' di Padova, Padova, Italy.

出版信息

JACC Basic Transl Sci. 2024 Sep 23;9(9):1123-1125. doi: 10.1016/j.jacbts.2024.06.009. eCollection 2024 Sep.

DOI:10.1016/j.jacbts.2024.06.009
PMID:39444929
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11494385/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ac3/11494385/05c70b0a978a/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ac3/11494385/2d1d72770b04/ga1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ac3/11494385/c316bfaf75c4/ga2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ac3/11494385/05c70b0a978a/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ac3/11494385/2d1d72770b04/ga1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ac3/11494385/c316bfaf75c4/ga2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ac3/11494385/05c70b0a978a/gr1.jpg

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本文引用的文献

1
A myocardial infarct border-zone-on-a-chip demonstrates distinct regulation of cardiac tissue function by an oxygen gradient.芯片中的心肌梗死边缘区可通过氧浓度梯度显著调节心脏组织功能。
Sci Adv. 2022 Dec 9;8(49):eabn7097. doi: 10.1126/sciadv.abn7097. Epub 2022 Dec 7.
2
Properties and Functions of Fibroblasts and Myofibroblasts in Myocardial Infarction.心肌梗死中成纤维细胞和肌成纤维细胞的特性和功能。
Cells. 2022 Apr 20;11(9):1386. doi: 10.3390/cells11091386.
3
Epigenetic State Changes Underlie Metabolic Switch in Mouse Post-Infarction Border Zone Cardiomyocytes.
表观遗传状态变化是小鼠心肌梗死后边缘区心肌细胞代谢转换的基础。
J Cardiovasc Dev Dis. 2021 Oct 22;8(11):134. doi: 10.3390/jcdd8110134.
4
miR-378a-3p inhibits ischemia/reperfusion-induced apoptosis in H9C2 cardiomyocytes by targeting TRIM55 via the DUSP1-JNK1/2 signaling pathway.miR-378a-3p 通过靶向 TRIM55 抑制 DUSP1-JNK1/2 信号通路抑制 H9C2 心肌细胞缺血/再灌注诱导的细胞凋亡。
Aging (Albany NY). 2020 May 28;12(10):8939-8952. doi: 10.18632/aging.103106.
5
Genetic and functional implications of an exonic TRIM55 variant in heart failure.心力衰竭中外显子TRIM55变异的遗传和功能意义
J Mol Cell Cardiol. 2020 Jan;138:222-233. doi: 10.1016/j.yjmcc.2019.12.008. Epub 2019 Dec 19.
6
Conditioned medium from hypoxic cells protects cardiomyocytes against ischemia.缺氧细胞条件培养液可保护心肌细胞免受缺血损伤。
Mol Cell Biochem. 2012 Apr;363(1-2):167-78. doi: 10.1007/s11010-011-1169-7. Epub 2011 Dec 8.
7
The FoxO family in cardiac function and dysfunction.FoxO 家族与心脏功能和功能障碍。
Annu Rev Physiol. 2010;72:81-94. doi: 10.1146/annurev-physiol-021909-135931.
8
Structural remodelling of cardiomyocytes in the border zone of infarcted rabbit heart.梗死兔心脏边缘区心肌细胞的结构重塑
Mol Cell Biochem. 2007 Aug;302(1-2):225-32. doi: 10.1007/s11010-007-9445-2. Epub 2007 Mar 27.
9
Extension of borderzone myocardium in postinfarction dilated cardiomyopathy.心肌梗死后扩张型心肌病中边缘区心肌的扩展
J Am Coll Cardiol. 2002 Sep 18;40(6):1160-7; discussion 1168-71. doi: 10.1016/s0735-1097(02)02121-6.