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通过综合生物信息学分析鉴定早期青光眼的序贯分子机制和关键生物标志物

Identification of Sequential Molecular Mechanisms and Key Biomarkers in Early Glaucoma by Integrated Bioinformatics Analysis.

作者信息

Huang Jingqiu, Chang Zhaohui, Deng Xizhi, Cai Shuncheng, Jiang Bin, Zeng Wen, Ke Min

机构信息

Department of Ophthalmology, Zhongnan Hospital of Wuhan University, Wuhan, Hubei, China.

出版信息

Mol Neurobiol. 2025 Apr;62(4):4952-4970. doi: 10.1007/s12035-024-04563-0. Epub 2024 Nov 4.

DOI:10.1007/s12035-024-04563-0
PMID:39495230
Abstract

Glaucoma is a neurodegenerative disease characterized by progressive optic nerve degeneration and retinal ganglion cell (RGC) loss. In early glaucoma, before obvious axon loss, highly organized pathological processes in RGCs occur sequentially, involving axons, dendrites and synaptic terminals. The optic nerve head (ONH) is the critical structure of early glaucomatous neurodegeneration. Taking advantage of high-throughput data from the ONH and the weighted gene coexpression network analysis (WGCNA) method, the current study aims to gain insight into the full scope of pathological events in early glaucoma and define their chronological sequence. The expression profiles of GSE26299, GSE110019, and GSE139605, which measure ONH gene expression in different glaucoma models, were downloaded from the Gene Expression Omnibus (GEO) database. In GSE26299, which uses 10.5-month-old DBA/2 J mice, WGCNA was utilized to construct a gene coexpression network, and the most significant modules of early (NOE), moderate (MOD) and severe (SEV) glaucoma were identified. The differentially expressed genes (DEGs) of GSE110019 and GSE139605 significantly overlapped with the correlated module of the MOD group, so the 3 gene sets were analyzed together. Pathway enrichment analysis via the GO, KEGG, and Reactome pathways was subsequently performed, followed by protein‒protein interaction (PPI) analysis to screen key genes associated with each stage. Several hub gene expression patterns were identified in a glucocorticoid-induced glaucoma (GIG) model via quantitative PCR and immunostaining. The pink module was positively correlated with the NOE group (r = 0.48, p = 4e-04) and negatively correlated with the glaucoma stage (r = -0.88, p = 3e-17). The genes in the pink module were enriched in the synaptic transmission and axonal transport pathways. The tan module was negatively correlated with the NOE group (r = -0.43, p = 0.002) and positively correlated with the glaucoma stage (r = 0.77, p = 7e-11). The genes in the tan module were associated with pathways such as tight junctions, retinol metabolism, and linoleic acid metabolism. The purple module was positively correlated with the MOD group (r = 0.64, p = 5e-07). The common genes among the purple module and the DEGs of the two other datasets were enriched in pathways related to mitotic cell division, cytokine activity, and the extracellular matrix (ECM). The hub genes identified by PPI included Nrn1, Cplx1, Timp1, and Cdk1. Quantitative PCR and immunostaining confirmed that Limk1 expression was increased in the ONH of GIG mice. In early glaucomatous neuropathy, intrinsic changes in RGCs precede the activation of glial cells and ECM remodeling. These latter events are common pathological changes observed in the ONH in both cats and mice. Our study may provide new targets for the early detection and treatment of glaucoma.

摘要

青光眼是一种神经退行性疾病,其特征为进行性视神经变性和视网膜神经节细胞(RGC)丧失。在早期青光眼阶段,在明显的轴突丧失之前,RGC中高度有序的病理过程会依次发生,涉及轴突、树突和突触终末。视神经乳头(ONH)是早期青光眼性神经变性的关键结构。利用来自ONH的高通量数据和加权基因共表达网络分析(WGCNA)方法,本研究旨在深入了解早期青光眼病理事件的全貌并确定其时间顺序。从基因表达综合数据库(GEO)下载了GSE26299、GSE110019和GSE139605的表达谱,这些表达谱测量了不同青光眼模型中ONH基因的表达。在使用10.5月龄DBA/2 J小鼠的GSE26299中,利用WGCNA构建基因共表达网络,并确定了早期(NOE)、中度(MOD)和重度(SEV)青光眼的最显著模块。GSE110019和GSE139605的差异表达基因(DEG)与MOD组的相关模块有显著重叠,因此将这3个基因集一起分析。随后通过GO、KEGG和Reactome通路进行通路富集分析,接着进行蛋白质-蛋白质相互作用(PPI)分析以筛选与每个阶段相关的关键基因。通过定量PCR和免疫染色在糖皮质激素诱导的青光眼(GIG)模型中鉴定了几种枢纽基因表达模式。粉色模块与NOE组呈正相关(r = 0.48,p = 4e - 04),与青光眼阶段呈负相关(r = -0.88,p = 3e - 17)。粉色模块中的基因在突触传递和轴突运输通路中富集。棕色模块与NOE组呈负相关(r = -0.43,p = 0.002),与青光眼阶段呈正相关(r = 0.77,p = 7e - 11)。棕色模块中的基因与紧密连接、视黄醇代谢和亚油酸代谢等通路相关。紫色模块与MOD组呈正相关(r = 0.64,p = 5e - 07)。紫色模块与其他两个数据集的DEG中的共同基因在与有丝分裂细胞分裂、细胞因子活性和细胞外基质(ECM)相关的通路中富集。通过PPI鉴定的枢纽基因包括Nrn1、Cplx1、Timp1和Cdk1。定量PCR和免疫染色证实GIG小鼠的ONH中Limk1表达增加。在早期青光眼性神经病变中,RGC的内在变化先于胶质细胞的激活和ECM重塑。后两者是在猫和小鼠的ONH中观察到的常见病理变化。我们的研究可能为青光眼的早期检测和治疗提供新的靶点。

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本文引用的文献

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Optic Nerve Head Gene Transcription Sequelae to a Single Elevated IOP Exposure Provides Insights Into Known Responses to Chronically Elevated IOP.单一升高的眼内压暴露对视神经头基因转录的后续影响为深入了解已知的慢性升高的眼内压反应提供了线索。
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Bax 促进青光眼期间视网膜神经节细胞树突退化。
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