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Gut microbiota modulates oligodendrocyte lineage cell response after traumatic brain injury.

作者信息

Shumilov Kirill, Friess Stuart

机构信息

Department of Neurosurgery, Virginia Commonwealth University, Richmond, VA, USA (Shumilov K).

Department of Pediatrics, Washington University in St. Louis School of Medicine, St. Louis, MO, USA (Friess S).

出版信息

Neural Regen Res. 2026 Jan 1;21(1):310-311. doi: 10.4103/NRR.NRR-D-24-01061. Epub 2024 Dec 7.

DOI:10.4103/NRR.NRR-D-24-01061
PMID:39665806
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12094551/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/46d3/12094551/a45daff6e385/NRR-21-310-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/46d3/12094551/a45daff6e385/NRR-21-310-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/46d3/12094551/a45daff6e385/NRR-21-310-g001.jpg

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本文引用的文献

1
Early depletion of gut microbiota shape oligodendrocyte response after traumatic brain injury.早期肠道微生物群耗竭可塑造创伤性脑损伤后的少突胶质细胞反应。
J Neuroinflammation. 2024 Jul 15;21(1):171. doi: 10.1186/s12974-024-03158-9.
2
T cell-mediated microglial activation triggers myelin pathology in a mouse model of amyloidosis.T 细胞介导的小胶质细胞激活触发淀粉样变性小鼠模型中的髓鞘病理学。
Nat Neurosci. 2024 Aug;27(8):1468-1474. doi: 10.1038/s41593-024-01682-8. Epub 2024 Jun 27.
3
Innate and Peripheral Immune Alterations after Traumatic Brain Injury Are Regulated in a Gut Microbiota-Dependent Manner in Mice.
创伤性脑损伤后先天和外周免疫改变受小鼠肠道微生物群调控。
J Neurotrauma. 2023 Apr;40(7-8):772-787. doi: 10.1089/neu.2022.0356. Epub 2023 Jan 13.
4
Brain-gut axis dysfunction in the pathogenesis of traumatic brain injury.脑-肠轴功能障碍在创伤性脑损伤发病机制中的作用。
J Clin Invest. 2021 Jun 15;131(12). doi: 10.1172/JCI143777.
5
Gut microbial dysbiosis after traumatic brain injury modulates the immune response and impairs neurogenesis.创伤性脑损伤后肠道微生物失调会调节免疫反应并损害神经发生。
Acta Neuropathol Commun. 2021 Mar 10;9(1):40. doi: 10.1186/s40478-021-01137-2.
6
Disease-specific oligodendrocyte lineage cells arise in multiple sclerosis.多发性硬化症中出现了特定于疾病的少突胶质细胞谱系细胞。
Nat Med. 2018 Dec;24(12):1837-1844. doi: 10.1038/s41591-018-0236-y. Epub 2018 Nov 12.
7
Regenerating CNS myelin - from mechanisms to experimental medicines.中枢神经系统髓鞘再生——从机制到实验药物。
Nat Rev Neurosci. 2017 Nov 16;18(12):753-769. doi: 10.1038/nrn.2017.136.
8
Diffuse traumatic brain injury in the mouse induces a transient proliferation of oligodendrocyte progenitor cells in injured white matter tracts.小鼠弥漫性创伤性脑损伤会诱导损伤白质束中少突胶质细胞祖细胞的短暂增殖。
Restor Neurol Neurosci. 2017;35(2):251-263. doi: 10.3233/RNN-160675.
9
Human Traumatic Brain Injury Results in Oligodendrocyte Death and Increases the Number of Oligodendrocyte Progenitor Cells.人类创伤性脑损伤导致少突胶质细胞死亡并增加少突胶质前体细胞数量。
J Neuropathol Exp Neurol. 2016 Jun;75(6):503-15. doi: 10.1093/jnen/nlw025. Epub 2016 Apr 22.
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Transl Psychiatry. 2016 Apr 5;6(4):e774. doi: 10.1038/tp.2016.42.