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Editorial: Advance in vascular anomalies of head and neck region: from bench to bedside.

作者信息

Wang Deming, Shen Yuchen, Xu Shancai, Cai Yu, Su Lixin, Yakes Wayne F, Fan Xindong

机构信息

Vascular Anomaly Center, Department of Interventional Therapy, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.

Department of Neurosurgery, The First Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.

出版信息

Front Neurol. 2024 Dec 20;15:1529950. doi: 10.3389/fneur.2024.1529950. eCollection 2024.

DOI:10.3389/fneur.2024.1529950
PMID:39758780
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11695843/
Abstract
摘要

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本文引用的文献

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EPHB4-RASA1-Mediated Negative Regulation of Ras-MAPK Signaling in the Vasculature: Implications for the Treatment of EPHB4- and RASA1-Related Vascular Anomalies in Humans.EPHB4-RASA1介导的血管中Ras-MAPK信号通路的负调控：对人类EPHB4和RASA1相关血管异常治疗的启示。
Pharmaceuticals (Basel). 2023 Jan 23;16(2):165. doi: 10.3390/ph16020165.
2
Hereditary Hemorrhagic Telangiectasia: Genetics, Pathophysiology, Diagnosis, and Management.遗传性出血性毛细血管扩张症：遗传学、病理生理学、诊断与管理
J Clin Med. 2022 Sep 5;11(17):5245. doi: 10.3390/jcm11175245.
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Intramuscular fast-flow vascular anomaly contains somatic MAP2K1 and KRAS mutations.肌内快血流血管异常包含体细胞 MAP2K1 和 KRAS 突变。
Angiogenesis. 2019 Nov;22(4):547-552. doi: 10.1007/s10456-019-09678-w. Epub 2019 Sep 5.
4
Endothelial Cells from Capillary Malformations Are Enriched for Somatic GNAQ Mutations.毛细血管畸形中的内皮细胞富含体细胞GNAQ突变。
Plast Reconstr Surg. 2016 Jan;137(1):77e-82e. doi: 10.1097/PRS.0000000000001868.
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Vascular dysmorphogenesis caused by an activating mutation in the receptor tyrosine kinase TIE2.受体酪氨酸激酶TIE2激活突变引起的血管发育异常。
Cell. 1996 Dec 27;87(7):1181-90. doi: 10.1016/s0092-8674(00)81814-0.