Cochran S D, Hammen C L
J Pers Soc Psychol. 1985 Jun;48(6):1562-71. doi: 10.1037//0022-3514.48.6.1562.
A brief review of tests of the attributional model of depression suggests that there is only weak or inconsistent support for the predicted causal ascriptions by depressed persons for negative events. Moreover, in very few studies have researchers actually tested the causal predictions of the model. Finally, because many of the studies were conducted with normal college students, the generality of the model as applied to different populations needs to be investigated. We addressed three questions: (a) the validity of the hypothesized independent and direction relation between each of the dimensions of internality, stability, controllability, intentionality, and globality and depression; (b) the causal relation between attributions and depression in a 2-month prospective study; and (c) evaluation of the model on two disparate samples: normal college students and elderly depressed outpatients ascribing causes for their personally stressful events. Causal modeling statistical procedures were applied to both the question of concurrent relations and causal relations between cognitions and depression. The results suggest minimal support for the attributional model: The dimensions were not each independently and directly associated with depression in the manner predicted by Abramson, Seligman, and Teasdale (1978), and the model that best fit the data was generally congruent for both the normal and clinical samples. In terms of direction of causality, the data were more consistent in indicating that depression causes cognitions than in indicating the reverse.
对抑郁症归因模型测试的简要回顾表明,抑郁症患者对负面事件的预测因果归因仅得到微弱或不一致的支持。此外,在极少数研究中,研究人员实际测试了该模型的因果预测。最后,由于许多研究是针对正常大学生进行的,因此需要研究该模型应用于不同人群时的普遍性。我们解决了三个问题:(a) 内在性、稳定性、可控性、意向性和全局性各维度与抑郁症之间假设的独立和方向关系的有效性;(b) 在一项为期2个月的前瞻性研究中归因与抑郁症之间的因果关系;(c) 在两个不同样本上对该模型进行评估:正常大学生和为自己的个人应激事件归因的老年抑郁症门诊患者。因果建模统计程序应用于认知与抑郁症之间的并发关系和因果关系问题。结果表明对归因模型的支持微乎其微:这些维度并非如阿布拉姆森、塞利格曼和蒂斯代尔(1978年)所预测的那样,各自独立且直接与抑郁症相关,并且最符合数据的模型在正常样本和临床样本中通常是一致的。就因果关系的方向而言,数据在表明抑郁症导致认知方面比表明相反情况更为一致。