Yi-Shueh Chen Fred, Yin-Kai Huang Dean
Department of Physical Medicine and Rehabilitation, Taipei Medical University Hospital, Taipei, Taiwan (R.O.C.).
J Musculoskelet Neuronal Interact. 2025 Jun 1;25(2):248-251.
We describe the case of a 41-year-old Asian man who was initially given a diagnosis of peroneal neuropathy but who later received a diagnosis of tenosynovitis of extensor digitorum longus (EDL). The patient initially presented with left lateral ankle numbness, pain, and decreased range of dorsiflexion after an 8-km walk. Peroneal neuropathy was first diagnosed on the basis of reduced compound muscle action potential (CMAP). Conversely, ankle ultrasound revealed normal peroneal nerve but considerable EDL tenosynovitis. Ultrasound-guided injection of triamcinolone and lidocaine into the tendon sheath was performed for pain relief. At 1-month follow-up, CMAP amplitude was restored, and ultrasound imaging revealed normal EDL structure without signs of tenosynovitis. This case serves as a reminder that clinicians must pay attention to multiple factors affecting CMAP, including tendinopathy and pain, to avoid misinterpretation.
我们描述了一名41岁亚洲男性的病例,该患者最初被诊断为腓总神经病变,但后来被诊断为趾长伸肌(EDL)腱鞘炎。患者最初在步行8公里后出现左侧外踝麻木、疼痛和背屈范围减小。最初根据复合肌肉动作电位(CMAP)降低诊断为腓总神经病变。相反,踝关节超声显示腓总神经正常,但存在明显的EDL腱鞘炎。为缓解疼痛,在超声引导下向腱鞘内注射了曲安奈德和利多卡因。在1个月的随访中,CMAP幅度恢复,超声成像显示EDL结构正常,无腱鞘炎迹象。该病例提醒临床医生必须注意影响CMAP的多种因素,包括肌腱病和疼痛,以避免误诊。