Sametoğlu Selim, Pelt Dirk H M, Bartels Meike
Max Planck Institute for Psycholinguistics, Nijmegen, Netherlands.
Vrije Universiteit Amsterdam, Amsterdam, Netherlands.
Behav Genet. 2025 Jun 21. doi: 10.1007/s10519-025-10224-2.
Meta-analyses report small to moderate effect sizes or inconsistent associations (usually around r = -0.10) between wellbeing (WB) and social media use (SMU) and between anxious-depressive symptoms (ADS) and SMU (also around r = 0.10). This study employs the classical twin design, utilizing data from 6492 individuals from the Netherlands Twin Register, including 3369 MZ twins (893 complete twin pairs, 1583 incomplete twin pairs) and 3123 DZ twins (445 complete, 2233 incomplete) to provide insights into the sources of overlap between WB/ADS and SMU. Both hedonic and eudaimonic WB scales were used. SMU was measured by (1) the time spent on different social media platforms (SMU), (2) the frequency of posting on social media (SMU), and (3) the number of social media accounts individuals have (SMU). Our results confirmed the low phenotypic correlations between WB and SMU (between r = -0.09 and 0.04) as well as between ADS and SMU (between r = 0.07 and 0.10). For SMU, heritability estimates between 32 and 72% were obtained. The small but significant phenotypic correlations between WB/ADS and the SMU phenotypes were mainly determined by genetic factors (in the range of 80-90%). For WB and SMU, genetic correlations were between -0.10 and -0.0, and for ADS and SMU genetic correlations were between 0.10 and 0.23. Genetic correlations implied limited but statistically significant sets of genes that affect WB/ADS and SMU levels. Overall, the results indicate that there is evidence that the small associations between WB/ADS and SMU are partly driven by overlapping genetic influences. We encourage researchers and experts to consider more personalized approaches when considering the association between WB and SMU, as well as understanding the reasons for individuals' observed SMU levels.
元分析报告称,幸福感(WB)与社交媒体使用(SMU)之间以及焦虑抑郁症状(ADS)与SMU之间的效应大小较小到中等或关联不一致(通常约为r = -0.10)(也约为r = 0.10)。本研究采用经典双生子设计,利用来自荷兰双生子登记处的6492人的数据,其中包括3369对同卵双胞胎(893对完整双胞胎,1583对不完整双胞胎)和3123对异卵双胞胎(445对完整,2233对不完整),以深入了解WB/ADS与SMU之间重叠的来源。同时使用了享乐主义和自我实现幸福感量表。SMU通过以下方式测量:(1)在不同社交媒体平台上花费的时间(SMU),(2)在社交媒体上发布内容的频率(SMU),以及(3)个人拥有的社交媒体账户数量(SMU)。我们的结果证实了WB与SMU之间(r在-0.09至0.04之间)以及ADS与SMU之间(r在0.07至0.10之间)的低表型相关性。对于SMU,获得的遗传度估计在32%至72%之间。WB/ADS与SMU表型之间虽小但显著的表型相关性主要由遗传因素决定(在80%至90%的范围内)。对于WB和SMU,遗传相关性在-0.10至-0.0之间,对于ADS和SMU,遗传相关性在0.10至0.23之间。遗传相关性意味着影响WB/ADS和SMU水平的基因集有限但具有统计学意义。总体而言,结果表明有证据表明WB/ADS与SMU之间的小关联部分是由重叠的遗传影响驱动的。我们鼓励研究人员和专家在考虑WB与SMU之间的关联以及理解个体观察到的SMU水平的原因时,考虑更个性化的方法。