Ernest J T
Am J Ophthalmol. 1977 Sep;84(3):341-4. doi: 10.1016/0002-9394(77)90676-6.
Systolic hypertension was induced in rhesus monkeys by obstructing the thoracic aorta and by ocular sympathectomy obtained with the retrobulbar injection of 6-hydroxydopamine. During the late phase of fluorescein angiography, fluorescein leaked from the choroidal circulation into the retina. The retinal vessels did not leak fluorescein. The effect was not permanent since fluorescein did not leak into the retina after the blood pressure returned to normal.
通过阻塞胸主动脉以及球后注射6-羟基多巴胺进行眼交感神经切除术，在恒河猴中诱发收缩期高血压。在荧光素血管造影的后期，荧光素从脉络膜循环渗漏到视网膜中。视网膜血管未渗漏荧光素。由于血压恢复正常后荧光素未渗漏到视网膜中，因此这种效应不是永久性的。
