新西兰小鼠自身免疫性疾病的研究。I. NZB、NZY和NZW品系以及NZB-NZW杂交种的遗传特征和自然史。 - Suppr

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Study of autoimmune disease in New Zealand mice. I. Genetic features and natural history of NZB, NZY and NZW strains and NZB-NZW hybrids.

作者信息

Braverman I M

出版信息

J Invest Dermatol. 1968 Jun;50(6):483-99. doi: 10.1038/jid.1968.79.

DOI:10.1038/jid.1968.79

PMID:4172399

Abstract

摘要

相似文献

Study of autoimmune disease in New Zealand mice. I. Genetic features and natural history of NZB, NZY and NZW strains and NZB-NZW hybrids.新西兰小鼠自身免疫性疾病的研究。I. NZB、NZY和NZW品系以及NZB-NZW杂交种的遗传特征和自然史。

J Invest Dermatol. 1968 Jun;50(6):483-99. doi: 10.1038/jid.1968.79.

Naturally occurring antibody response to DNA is associated with the response to retroviral gp70 in autoimmune New Zealand mice.在自身免疫性新西兰小鼠中，对DNA的天然抗体反应与对逆转录病毒糖蛋白70的反应相关。

Arthritis Rheum. 1986 Feb;29(2):242-50. doi: 10.1002/art.1780290213.

Study of autoimmune disease in New Zealand mice. II. Immunologic and parabiotic studies of NZB-NZW F1 hybrids.新西兰小鼠自身免疫性疾病的研究。II. NZB-NZW F1 杂交种的免疫学和联体生活研究。

J Invest Dermatol. 1967 Nov;49(5):469-73. doi: 10.1038/jid.1967.167.

[NZB mice].[新西兰黑鼠]

Pathol Biol. 1968 Oct;16(19):885-92.

Suppression of autoimmune disease in New Zealand mice associated with infection with malaria. II. NZB mice.与疟疾感染相关的新西兰小鼠自身免疫疾病的抑制。II. NZB小鼠。

Clin Exp Immunol. 1970 Dec;7(6):805-15.

Ability of the xid gene to prevent autoimmunity in (NZB X NZW)F1 mice during the course of their natural history, after polyclonal stimulation, or following immunization with DNA.xid基因在（新西兰黑鼠×新西兰白鼠）F1小鼠自然病程中、多克隆刺激后或DNA免疫后预防自身免疫的能力。

J Clin Invest. 1982 Sep;70(3):587-97. doi: 10.1172/jci110651.

Differences in expression of lupus nephritis in New Zealand mixed H-2z homozygous inbred strains of mice derived from New Zealand black and New Zealand white mice. Origins and initial characterization.源自新西兰黑鼠和新西兰白鼠的新西兰混合H-2z纯合近交系小鼠狼疮性肾炎的表达差异。起源与初步特征。

Lab Invest. 1993 Apr;68(4):419-26.

Auto-immune disease in (NZB x CFW) F1 mice.（新西兰黑鼠×CFW 小鼠）F1 代小鼠的自身免疫性疾病

J Pathol Bacteriol. 1968 Apr;95(2):391-403. doi: 10.1002/path.1700950206.

The genetic contribution of the NZB mouse to the renal disease of the NZB x NZW hybrid.NZB小鼠对NZB×NZW杂交种肾脏疾病的遗传贡献。

Clin Exp Immunol. 1977 May;28(2):352-8.

The disease of the NZB mouse. Examination of exchange ovum transplantation derived NZB and CFW mice.新西兰黑鼠的疾病。对交换卵移植获得的新西兰黑鼠和CFW鼠的检查。

Clin Exp Immunol. 1972 Mar;10(3):493-514.

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Increased Goodpasture antigen-binding protein expression induces type IV collagen disorganization and deposit of immunoglobulin A in glomerular basement membrane.Goodpasture抗原结合蛋白表达增加会导致IV型胶原紊乱以及免疫球蛋白A在肾小球基底膜沉积。

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Production of congenic mouse strains carrying genomic intervals containing SLE-susceptibility genes derived from the SLE-prone NZM2410 strain.携带源自易患系统性红斑狼疮的新西兰黑鼠2410品系的系统性红斑狼疮易感基因的基因组区间的同源近交系小鼠品系的培育。

Mamm Genome. 1996 May;7(5):335-9. doi: 10.1007/s003359900098.

Genetic studies in NZB mice. V. Recombinant inbred lines demonstrate that separate genes control autoimmune phenotype.新西兰黑鼠的遗传学研究。V. 重组近交系表明不同基因控制自身免疫表型。

J Exp Med. 1981 May 1;153(5):1187-97. doi: 10.1084/jem.153.5.1187.

T cell abnormalities in NZB mice occur independently of autoantibody production.NZB小鼠的T细胞异常独立于自身抗体产生而发生。

J Exp Med. 1981 Feb 1;153(2):221-34. doi: 10.1084/jem.153.2.221.

Clin Exp Immunol. 1971 Mar;8(3):501-9.

The pathogenesis of autoimmunity in New Zealand mice. 3. Factors influencing the formation of anti-nucleic acid antibodies.新西兰小鼠自身免疫的发病机制。3. 影响抗核酸抗体形成的因素。

Immunology. 1971 Apr;20(4):523-31.

The genetic basis of autoimmunity in NZB mice studied by progeny-testing.通过子代检测研究NZB小鼠自身免疫的遗传基础。

Clin Exp Immunol. 1971 Mar;8(3):479-90.

In vitro studies on strain-dependent production of thymus-specific autoantibodies.胸腺特异性自身抗体菌株依赖性产生的体外研究。

J Exp Med. 1973 Jul 1;138(1):16-32. doi: 10.1084/jem.138.1.16.

The contribution of NZW genes to lupus-like disease in (NZB x NZW)F1 mice.新西兰白兔（NZW）基因对（新西兰黑兔×新西兰白兔）F1代小鼠狼疮样疾病的影响。

J Exp Med. 1987 May 1;165(5):1237-51. doi: 10.1084/jem.165.5.1237.

Influence of diet on survival of mice.饮食对小鼠生存的影响。

Proc Natl Acad Sci U S A. 1976 Apr;73(4):1279-83. doi: 10.1073/pnas.73.4.1279.

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