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Quantitative phagocytosis by neutrophils. II. Release of the C5-cleaving enzyme and inhibition of phagocytosis by rheumatoid factor.

作者信息

Ward P A, Zvaifler N J

出版信息

J Immunol. 1973 Dec;111(6):1777-82.

PMID:4201556
Abstract
摘要

相似文献

1
Quantitative phagocytosis by neutrophils. II. Release of the C5-cleaving enzyme and inhibition of phagocytosis by rheumatoid factor.中性粒细胞的定量吞噬作用。II. C5裂解酶的释放及类风湿因子对吞噬作用的抑制
J Immunol. 1973 Dec;111(6):1777-82.
2
Neutrophil and rheumatoid factor-Immunoglobulin G insoluble complex interactions: phagocytosis and sequelae.中性粒细胞与类风湿因子-免疫球蛋白G不溶性复合物的相互作用：吞噬作用及后遗症
J Rheumatol. 1976 Jun;3(2):109-17.
3
The immunologic release of constituents from neutrophil leukocytes. I. The role of antibody and complement on nonphagocytosable surfaces or phagocytosable particles.中性粒细胞中成分的免疫释放。I. 抗体和补体在不可吞噬表面或可吞噬颗粒上的作用。
J Immunol. 1971 Dec;107(6):1535-46.
4
Mechanisms of lysosomal enzyme release from leukocytes exposed to immune complexes and other particles.白细胞暴露于免疫复合物及其他颗粒时溶酶体酶释放的机制。
J Exp Med. 1971 Sep 1;134(3 Pt 2):149s-165s.
5
The ability of chemotactic factors to induce lysosomal enzyme release. I. The characteristics of the release, the importance of surfaces and the relation of enzyme release to chemotactic responsiveness.趋化因子诱导溶酶体酶释放的能力。I. 释放的特征、表面的重要性以及酶释放与趋化反应性的关系。
J Immunol. 1974 Jun;112(6):2047-54.
6
Neutrophilic leukocytes in immunologic reactions: evidence for the selective release of lysosomal constituents.免疫反应中的嗜中性白细胞：溶酶体成分选择性释放的证据。
J Immunol. 1972 Feb;108(2):310-7.
7
C5 chemotactic fragments produced by an enzyme in lysosomal granules of neutrophils.由中性粒细胞溶酶体颗粒中的一种酶产生的C5趋化片段。
J Immunol. 1970 Mar;104(3):535-43.
8
The incidence of precipitating activity in monoclonal proteins.单克隆蛋白中沉淀活性的发生率。
Clin Exp Immunol. 1971 Feb;8(2):263-9.
9
Differentiation of human IgM subclasses by the ability to interact with a factor resembling the first component of complement.通过与类似补体第一成分的因子相互作用的能力对人IgM亚类进行区分。
J Immunol. 1969 Sep;103(3):607-12.
10
Waldenström's macroglobulinaemia with anti-IgG activity: a series of five cases.伴有抗IgG活性的华氏巨球蛋白血症：5例病例系列
Clin Exp Immunol. 1970 Apr;6(4):531-45.

引用本文的文献

1
Granulocytes (the red, white, and blue) in hypersensitivity reactions: A review.粒细胞（红、白、蓝）在超敏反应中的作用：综述。
Inflammation. 1976 Mar;1(2):127-41. doi: 10.1007/BF00917524.
2
Modulation of the inflammatory response by products released from human polymorphonuclear leukocytes during phagocytosis : Generation and inactivation of the chemotactic factor C5a.人多形核白细胞吞噬过程中释放产物对炎症反应的调节：趋化因子 C5a 的产生和失活。
Inflammation. 1975 Mar;1(1):23-39. doi: 10.1007/BF00918057.
3
The complement system.补体系统。
Cell Tissue Res. 2011 Jan;343(1):227-35. doi: 10.1007/s00441-010-1034-0. Epub 2010 Sep 14.
4
In vitro and in vivo stimulation of rat neutrophils and alveolar macrophages by immune complexes. Production of O-2 and H2O2.免疫复合物对大鼠中性粒细胞和肺泡巨噬细胞的体外及体内刺激。超氧阴离子（O-2）和过氧化氢（H2O2）的产生。
Am J Pathol. 1983 Mar;110(3):297-309.
5
Pathopysiological aspects of immune complex diseases. Part II. Phagocytosis, exocytosis, and pathogenic depositions.免疫复合物疾病的病理生理学方面。第二部分。吞噬作用、胞吐作用和致病性沉积。
Klin Wochenschr. 1980 Jun 16;58(12):593-605. doi: 10.1007/BF01477835.
6
Leukotaxis and leukotactic disorders. A review.白细胞趋化作用与白细胞趋化性疾病。综述
Am J Pathol. 1974 Dec;77(3):520-38.
7
Studies on rheumatoid factor: I. The effect of rheumatoid factor on the clearance of preformed immune complexes in mice.类风湿因子的研究：I. 类风湿因子对小鼠体内预先形成的免疫复合物清除的影响。
Clin Exp Immunol. 1986 Dec;66(3):648-53.
8
Non-lethal complement-membrane attack on human neutrophils: transient cell swelling and metabolic depletion.非致死性补体对人中性粒细胞的膜攻击：短暂的细胞肿胀和代谢耗竭。
Immunology. 1988 Jan;63(1):71-7.
9
Generation of superoxide by immunologically stimulated normal human neutrophils and possible modulation by intracellular and extracellular SOD and rheumatoid factors.免疫刺激的正常人中性粒细胞产生超氧化物以及细胞内和细胞外超氧化物歧化酶和类风湿因子的可能调节作用。
Inflammation. 1987 Jun;11(2):143-51. doi: 10.1007/BF00916016.
10
Enhanced uptake by guinea-pig macrophages of radio-iodinated human aggregated immunoglobulin G in the presence of sera from rheumatoid patients with cutaneous vasculitis.在患有皮肤血管炎的类风湿患者血清存在的情况下，豚鼠巨噬细胞对放射性碘化人聚合免疫球蛋白G的摄取增强。
Clin Exp Immunol. 1975 Feb;19(2):267-80.