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Interaction of cells with immune complexes: adherence, release of constituents, and tissue injury.

作者信息

Henson P M

出版信息

J Exp Med. 1971 Sep 1;134(3 Pt 2):114s-135s.

PMID:4255113
Abstract
摘要

相似文献

1
Interaction of cells with immune complexes: adherence, release of constituents, and tissue injury.细胞与免疫复合物的相互作用:黏附、成分释放及组织损伤。
J Exp Med. 1971 Sep 1;134(3 Pt 2):114s-135s.
2
The immunologic release of constituents from neutrophil leukocytes. II. Mechanisms of release during phagocytosis, and adherence to nonphagocytosable surfaces.嗜中性白细胞成分的免疫释放。II. 吞噬作用期间的释放机制,以及对不可吞噬表面的黏附。
J Immunol. 1971 Dec;107(6):1547-57.
3
The immunologic release of constituents from neutrophil leukocytes. I. The role of antibody and complement on nonphagocytosable surfaces or phagocytosable particles.中性粒细胞中成分的免疫释放。I. 抗体和补体在不可吞噬表面或可吞噬颗粒上的作用。
J Immunol. 1971 Dec;107(6):1535-46.
4
Biopolymer membrane: a model system for the study of the neutrophilic leukocyte response to immune complexes.生物聚合物膜:用于研究嗜中性白细胞对免疫复合物反应的模型系统。
J Immunol. 1971 Aug;107(2):344-52.
5
Mechanisms of lysosomal enzyme release from leukocytes exposed to immune complexes and other particles.白细胞暴露于免疫复合物及其他颗粒时溶酶体酶释放的机制。
J Exp Med. 1971 Sep 1;134(3 Pt 2):149s-165s.
6
Neutrophilic leukocytes in immunologic reactions: evidence for the selective release of lysosomal constituents.免疫反应中的嗜中性白细胞:溶酶体成分选择性释放的证据。
J Immunol. 1972 Feb;108(2):310-7.
7
The pathogenicity of antigen-antibody complexes.
Pathol Annu. 1970;5:355-79.
8
[Vascular and renal lesions caused by antigen-antibody complexes. Pathomechanism of the serum disease type].[抗原-抗体复合物引起的血管和肾脏病变。血清病型的发病机制]
Med Welt. 1971 Jun 12;24:988-95.
9
Characterization of cationic protein-bearing granules of polymorphonuclear leukocytes.多形核白细胞含阳离子蛋白颗粒的特性分析。
Lab Invest. 1971 Mar;24(3):229-36.
10
Immunologic tissue injury mediated by neutrophilic leukocytes.由嗜中性白细胞介导的免疫性组织损伤。
Adv Immunol. 1968;9:97-162. doi: 10.1016/s0065-2776(08)60442-3.

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Membrane-cytoskeletal crosstalk mediated by myosin-I regulates adhesion turnover during phagocytosis.肌球蛋白-I 介导的膜细胞骨架相互作用调节吞噬作用过程中的黏附周转。
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Systems Level Dissection of Candida Recognition by Dectins: A Matter of Fungal Morphology and Site of Infection.通过脱(β-1,3)-葡聚糖识别受体对念珠菌进行系统水平剖析:真菌形态与感染部位的问题
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Ultrastructural alterations during ATP-induced secretion of lysosomal enzymes from rabbit polymorphonuclear leukocytes.
兔多形核白细胞在 ATP 诱导的溶酶体酶分泌过程中的超微结构改变。
Inflammation. 1975 Mar;1(1):85-91. doi: 10.1007/BF00918061.
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Fcgamma receptor cross-linking stimulates cell proliferation of macrophages via the ERK pathway.Fcγ 受体交联通过 ERK 通路刺激巨噬细胞的增殖。
J Biol Chem. 2010 Feb 5;285(6):4232-4242. doi: 10.1074/jbc.M109.037168. Epub 2009 Dec 8.
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Absence of C1q leads to less neuropathology in transgenic mouse models of Alzheimer's disease.在阿尔茨海默病转基因小鼠模型中,C1q的缺失导致较少的神经病理学变化。
J Neurosci. 2004 Jul 21;24(29):6457-65. doi: 10.1523/JNEUROSCI.0901-04.2004.
6
The possible role of neutrophil proteinases in damage to articular cartilage. 1978.中性粒细胞蛋白酶在关节软骨损伤中的可能作用。1978年。
Agents Actions. 1994 Dec;43(3-4):194-200; discussion 200-1. doi: 10.1007/BF01986688.
7
Neutrophil expression of tumour necrosis factor receptors (TNF-R) and of activation markers (CD11b, CD43, CD63) in rheumatoid arthritis.类风湿关节炎中肿瘤坏死因子受体(TNF-R)及活化标志物(CD11b、CD43、CD63)的中性粒细胞表达
Clin Exp Immunol. 1995 Jul;101(1):25-32. doi: 10.1111/j.1365-2249.1995.tb02272.x.
8
Leukocytes in chemotactic-fragment-induced lung inflammation. Vascular emigration and alveolar surface migration.趋化因子诱导的肺部炎症中的白细胞。血管迁移和肺泡表面迁移。
Am J Pathol. 1980 Nov;101(2):283-302.
9
Characteristics of N-formyl-methionyl-leucyl-phenylalanine as an inducer of lysosomal enzyme release from human neutrophils.N-甲酰甲硫氨酰-亮氨酰-苯丙氨酸作为人中性粒细胞溶酶体酶释放诱导剂的特性
Inflammation. 1980 Mar;4(1):73-88. doi: 10.1007/BF00914105.
10
Inhibition of neutrophil migration by aggregated immunoglobulin attached to micropore membranes.附着于微孔膜的聚集免疫球蛋白对中性粒细胞迁移的抑制作用。
Immunology. 1980 Apr;39(4):529-34.