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The determination of the oxidation-reduction states of ubiquinone (coenzyme Q) in rat-liver mitochondria.

作者信息

Redfearn E R, Whittaker P A

出版信息

Biochim Biophys Acta. 1966 May 5;118(2):413-8. doi: 10.1016/s0926-6593(66)80050-4.

DOI:10.1016/s0926-6593(66)80050-4
PMID:4289837
Abstract
摘要

相似文献

1
The determination of the oxidation-reduction states of ubiquinone (coenzyme Q) in rat-liver mitochondria.
Biochim Biophys Acta. 1966 May 5;118(2):413-8. doi: 10.1016/s0926-6593(66)80050-4.
2
Oxidation-reduction status of coenzyme Q in rat liver microsomes.大鼠肝脏微粒体中辅酶Q的氧化还原状态
Indian J Biochem. 1965 Jun;2(2):80-4.
3
Oxidation-reduction levels of ubiquinone (coenzyme Q) in different metabolic states of rat liver mitochondria.大鼠肝脏线粒体不同代谢状态下泛醌（辅酶Q）的氧化还原水平
Biochem Biophys Res Commun. 1960 Dec;3:650-3. doi: 10.1016/0006-291x(60)90080-2.
4
Proton translocation coupled to quinone reduction by reduced nicotinamide--adenine dinucleotide in rat liver and ox heart mitochondria.大鼠肝脏和牛心脏线粒体中质子转运与还原型烟酰胺腺嘌呤二核苷酸介导的醌还原相偶联。
Biochem J. 1972 Dec;130(4):1029-44. doi: 10.1042/bj1301029.
5
Ubiquinone in proline oxidation.脯氨酸氧化中的泛醌。
Arch Int Pharmacodyn Ther. 1965 Dec;158(2):209-15.
6
Studies on reduced and oxidized coenzyme Q (ubiquinones). II. The determination of oxidation-reduction levels of coenzyme Q in mitochondria, microsomes and plasma by high-performance liquid chromatography.还原型和氧化型辅酶Q（泛醌）的研究。II. 用高效液相色谱法测定线粒体、微粒体和血浆中辅酶Q的氧化还原水平。
Biochim Biophys Acta. 1982 Feb 17;679(2):308-14. doi: 10.1016/0005-2728(82)90301-2.
7
Inhibition of mitochondrial beta-oxidation in the heart by increased redox state of the ubiquinone pool.
Biochem Soc Trans. 1996 Aug;24(3):457S. doi: 10.1042/bst024457s.
8
Proton translocation by rat liver mitochondria with quinones as terminal acceptors.以醌类作为末端受体时大鼠肝脏线粒体的质子转运
Biochem J. 1972 Sep;129(3):47P-48P. doi: 10.1042/bj1290047pb.
9
In vitro effects of glucocorticoid on mitochondrial energy metabolism.糖皮质激素对线粒体能量代谢的体外作用。
Biochim Biophys Acta. 1991 Jun 17;1058(2):152-60. doi: 10.1016/s0005-2728(05)80232-4.
10
The kinetics of changes in the redox state of ubiquinone on the transition from state 4 to state 3 in rat-liver mitochondria.大鼠肝脏线粒体从状态4转变为状态3时泛醌氧化还原状态变化的动力学。
Biochim Biophys Acta. 1970 Mar 3;197(2):113-6. doi: 10.1016/0005-2728(70)90021-6.

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Differential protein acetylation assists import of excess SOD2 into mitochondria and mediates SOD2 aggregation associated with cardiac hypertrophy in the murine SOD2-tg heart.差异性蛋白质乙酰化作用有助于将过量的超氧化物歧化酶2（SOD2）导入线粒体，并介导与小鼠SOD2转基因心脏中心脏肥大相关的SOD2聚集。
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BCNU-induced gR2 defect mediates S-glutathionylation of Complex I and respiratory uncoupling in myocardium.BCNU 诱导的 gR2 缺陷介导心肌细胞复合体 I 的 S-谷胱甘肽化和呼吸解偶联。
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Protection of rat skeletal muscle fibers by either L-carnitine or coenzyme Q10 against statins toxicity mediated by mitochondrial reactive oxygen generation.左旋肉碱或辅酶 Q10 对他汀类药物介导的线粒体活性氧生成所致大鼠骨骼肌纤维毒性的保护作用。
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Design and use of peptide-based antibodies decreasing superoxide production by mitochondrial complex I and complex II.设计和使用基于肽的抗体来减少线粒体复合物 I 和复合物 II 的超氧化物产生。
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Peptide-based antibodies against glutathione-binding domains suppress superoxide production mediated by mitochondrial complex I.针对谷胱甘肽结合域的肽类抗体抑制由线粒体复合物 I 介导的超氧化物产生。
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Mass spectrometry profiles superoxide-induced intramolecular disulfide in the FMN-binding subunit of mitochondrial Complex I.质谱分析线粒体复合体I的黄素单核苷酸结合亚基中超氧化物诱导的分子内二硫键。
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