Early step in Sendai virus-mediated rescue of Rous sarcoma virus from virogenic mammalian cells.
作者信息
Svoboda J, Machala O
出版信息
J Virol. 1968 Sep;2(9):951-2. doi: 10.1128/JVI.2.9.951-952.1968.
Abstract
摘要
相似文献
1
Early step in Sendai virus-mediated rescue of Rous sarcoma virus from virogenic mammalian cells.
J Virol. 1968 Sep;2(9):951-2. doi: 10.1128/JVI.2.9.951-952.1968.
2
Rescue of Rous sarcoma virus from virogenic mammalian cells associated with chicken cells and treated with Sendai virus.
J Gen Virol. 1969 Jun;4(4):523-9. doi: 10.1099/0022-1317-4-4-523.
3
A full expression of the genome of Rous sarcoma virus in heterokaryons formed after fusion of virogenic mammalian cells and chicken fibroblasts.
J Gen Virol. 1970 Sep;8(3):219-29. doi: 10.1099/0022-1317-8-3-219.
4
Influence of UV-inactivated HVJ on RSV rescue in cultures of different virogenic mammalian cells mixed with normal sensitive chicken cells.
Folia Biol (Praha). 1970;16(2):137-40.
5
Rescue of rous sarcoma virus from rous sarcoma virus-transformed mammalian cells.
J Virol. 1972 Jul;10(1):153-6. doi: 10.1128/JVI.10.1.153-156.1972.
6
Rescue of Rous sarcoma virus in mixed cultures of virogenic mammalian and chicken cells, treated and untreated with Sendai virus and detected by focus assay.
J Gen Virol. 1968 May;2(3):461-4. doi: 10.1099/0022-1317-2-3-461.
7
Complementation rescue of Rous sarcoma virus from transformed mammalian cells by polyethylene glycol-mediated cell fusion.
J Virol. 1977 Jul;23(1):133-41. doi: 10.1128/JVI.23.1.133-141.1977.
8
Persistence of Rous sarcoma virus in transformed non-permissive cells: mechanism of virus induction by association with permissive cells in the absence of Sendai virus.
Int J Cancer. 1972 Jan 15;9(1):150-61. doi: 10.1002/ijc.2910090118.
9
The heritable nature of the factor in chicken cells which acts as a helper virus for Rous sarcoma virus.
Virology. 1971 Aug;45(2):508-15. doi: 10.1016/0042-6822(71)90351-5.
10
Persistence of Rous sarcoma virus in transformed nonpermissive cells: relationship between virus induction by association with permissive cells and gs antigen content of transformed cells.
Virology. 1971 Jul;45(1):309-12. doi: 10.1016/0042-6822(71)90139-5.
本文引用的文献
1
Studies on herpes simplex virus. I. The stability and preservation of egg-adapted herpes simplex virus.
J Bacteriol. 1951 Mar;61(3):253-8. doi: 10.1128/jb.61.3.253-258.1951.
2
RELATIONSHIP BETWEEN THE ENVELOPE AND THE INFECTIVITY OF HERPES SIMPLEX VIRUS.
Proc Soc Exp Biol Med. 1964 Mar;115:814-6. doi: 10.3181/00379727-115-29045.
3
SOME SEROLOGICAL PROPERTIES OF HERPES VIRUS PARTICLES STUDIED WITH THE ELECTRON MICROSCOPE.
Virology. 1963 Sep;21:100-11. doi: 10.1016/0042-6822(63)90308-8.
4
Virologic and clinical observations on cytomegalic inclusion disease.
N Engl J Med. 1962 Jun 14;266:1233-44. doi: 10.1056/NEJM196206142662401.
5
The denaturation of lipid-protein complexes as a cause of damage by freezing.
Proc R Soc Lond B Biol Sci. 1957 Dec 17;147(929):427-33. doi: 10.1098/rspb.1957.0062.
6
Mechanics of freezing in living cells and tissues.
Science. 1956 Sep 21;124(3221):515-21. doi: 10.1126/science.124.3221.515.
7
Tissue freezing. A theory for injury and survival.
Cryobiology. 1965 Nov-Dec;2(3):99-108. doi: 10.1016/s0011-2240(65)80094-3.
8
Correlation between infectivity and physical virus particles in human cytomegalovirus.
J Bacteriol. 1966 Nov;92(5):1555-61. doi: 10.1128/jb.92.5.1555-1561.1966.
Zotero 插件