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On the slowness of action of tricyclic antidepressant drugs.

作者信息

Oswald I, Brezinova V, Dunleavy D L

出版信息

Br J Psychiatry. 1972 Jun;120(559):673-7. doi: 10.1192/bjp.120.559.673.

DOI:10.1192/bjp.120.559.673
PMID:4339631
Abstract
摘要

相似文献

1
On the slowness of action of tricyclic antidepressant drugs.论三环类抗抑郁药的作用迟缓性。
Br J Psychiatry. 1972 Jun;120(559):673-7. doi: 10.1192/bjp.120.559.673.
2
Changes during weeks in effects of tricyclic drugs on the human sleeping brain.
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3
Evidence for REM sleep deprivation as the mechanism of action of antidepressant drugs.快速眼动睡眠剥夺作为抗抑郁药物作用机制的证据。
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4
Depression and MHPG excretion. Response to dextroamphetamine and tricyclic antidepressants.抑郁症与3-甲氧基-4-羟基苯乙二醇（MHPG）排泄。对右旋苯丙胺和三环类抗抑郁药的反应。
Arch Gen Psychiatry. 1972 Mar;26(3):246-51. doi: 10.1001/archpsyc.1972.01750210054011.
5
Theoretical view of REM sleep function: maintenance of catecholamine systems in the central nervous system.
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6
Behavioural despair in rats: a new model sensitive to antidepressant treatments.大鼠的行为绝望：一种对抗抑郁治疗敏感的新模型。
Eur J Pharmacol. 1978 Feb 15;47(4):379-91. doi: 10.1016/0014-2999(78)90118-8.
7
In vivo studies on central noradrenergic synaptic mechanisms after acute and chronic antidepressant drug treatment: biochemical and behavioral comparison.
J Pharmacol Exp Ther. 1982 Oct;223(1):227-34.
8
Effects of imipramine on enuretic frequency and sleep stages.丙咪嗪对遗尿频率和睡眠阶段的影响。
Pediatrics. 1977 Oct;60(4):431-6.
9
Ketamine plus imipramine treatment induces antidepressant-like behavior and increases CREB and BDNF protein levels and PKA and PKC phosphorylation in rat brain.氯胺酮联合丙咪嗪治疗可诱导大鼠产生抗抑郁样行为，并增加大脑中 CREB 和 BDNF 蛋白的水平以及 PKA 和 PKC 的磷酸化。
Behav Brain Res. 2011 Aug 1;221(1):166-71. doi: 10.1016/j.bbr.2011.02.024. Epub 2011 Mar 21.
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Studies on the participation of the dopaminergic system in the central effects of chronically administered antidepressant drugs.多巴胺能系统参与长期服用抗抑郁药物的中枢效应的研究。
Pol J Pharmacol Pharm. 1983;35(2):115-26.

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Neuroinflammation as an etiological trigger for depression comorbid with inflammatory bowel disease.神经炎症作为并发炎症性肠病的抑郁症的病因触发因素。
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Blues in the Brain and Beyond: Molecular Bases of Major Depressive Disorder and Relative Pharmacological and Non-Pharmacological Treatments.
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Mesolimbic dopamine neurons in the brain reward circuit mediate susceptibility to social defeat and antidepressant action.大脑奖励回路中的中脑边缘多巴胺神经元介导对社交挫败的易感性和抗抑郁作用。
J Neurosci. 2010 Dec 8;30(49):16453-8. doi: 10.1523/JNEUROSCI.3177-10.2010.
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Chronic administration of imipramine and citalopram alters the expression of NMDA receptor subunit mRNAs in mouse brain. A quantitative in situ hybridization study.慢性给予丙咪嗪和西酞普兰会改变小鼠脑中NMDA受体亚基mRNA的表达。一项定量原位杂交研究。
J Mol Neurosci. 1998 Jun;10(3):219-33. doi: 10.1007/BF02761776.
7
Biochemistry and pharmacology of reversible inhibitors of MAO-A agents: focus on moclobemide.单胺氧化酶A（MAO-A）可逆性抑制剂的生物化学与药理学：聚焦吗氯贝胺
J Psychiatry Neurosci. 1993 Nov;18(5):214-25.
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Beta-receptor responsiveness after desipramine treatment.去甲丙咪嗪治疗后的β受体反应性。
Psychopharmacology (Berl). 1993;110(1-2):37-44. doi: 10.1007/BF02246948.
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Prevention of the serotonin syndrome in rats by repeated administration of monoamine oxidase inhibitors but not tricyclic antidepressants.通过重复给予单胺氧化酶抑制剂而非三环类抗抑郁药预防大鼠血清素综合征。
Psychopharmacology (Berl). 1982;77(3):205-11. doi: 10.1007/BF00464567.
10
Changes in action of tricyclic and tetracyclic antidepressants: desipramine and mianserin, on avoidance behavior in the course of the chronic treatment.三环和四环抗抑郁药（地昔帕明和米安色林）在长期治疗过程中对回避行为作用的变化。
Psychopharmacology (Berl). 1981;73(3):265-8. doi: 10.1007/BF00422415.