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Effects of anti-inflammatory drugs on prostaglandin biosynthesis.

作者信息

Flower R, Gryglewski R, Herbaczyńska-Cedro K, Vane J R

出版信息

Nat New Biol. 1972 Jul 26;238(82):104-6. doi: 10.1038/newbio238104a0.

DOI:10.1038/newbio238104a0
PMID:4505422
Abstract
摘要

相似文献

1
Effects of anti-inflammatory drugs on prostaglandin biosynthesis.抗炎药物对前列腺素生物合成的影响。
Nat New Biol. 1972 Jul 26;238(82):104-6. doi: 10.1038/newbio238104a0.
2
The influence of anti-inflammatory and immunosuppressant drugs on Nystatin induced oedema.抗炎和免疫抑制药物对制霉菌素诱导水肿的影响。
Pharmacology. 1971;5(4):215-24. doi: 10.1159/000136193.
3
A rapid spectrophotometric assay for prostaglandin synthetase: application to the study of non-steroidal antiinflammatory agents.一种用于前列腺素合成酶的快速分光光度测定法:应用于非甾体抗炎药的研究。
Prostaglandins. 1972 Sep;2(3):169-84. doi: 10.1016/s0090-6980(72)80021-2.
4
In vitro effect of anti-inflammatory agents on phagocytosis and bacterial killing by human neutrophilic leukocytes.
Acta Pharmacol Toxicol (Copenh). 1972;31(3):226-37. doi: 10.1111/j.1600-0773.1972.tb00717.x.
5
Platelet aggregation induced by arachidonic acid is accompanied by release of potential inflammatory mediators distinct from PGE2 and PGF2.花生四烯酸诱导的血小板聚集伴随着不同于前列腺素E2和前列腺素F2的潜在炎症介质的释放。
Nat New Biol. 1973 Jul 25;244(134):114-6. doi: 10.1038/newbio244114a0.
6
A comparison of the inhibitory activity of compounds on ocular prostaglandin biosynthesis.化合物对眼部前列腺素生物合成的抑制活性比较。
Invest Ophthalmol. 1974 Dec;13(12):967-72.
7
Anti-inflammatory drugs in experimental atherosclerosis. I. Relative potencies for inhibiting plaque formation.
Atherosclerosis. 1973 May-Jun;17(3):515-22. doi: 10.1016/0021-9150(73)90041-5.
8
Structural requirements for time-dependent inhibition of prostaglandin biosynthesis by anti-inflammatory drugs.抗炎药物对前列腺素生物合成的时间依赖性抑制的结构要求。
Proc Natl Acad Sci U S A. 1975 Dec;72(12):4863-5. doi: 10.1073/pnas.72.12.4863.
9
A method for differentiating nonspecific irritants from anti-inflammatory agents using the carrageenin abscess test.
Proc Soc Exp Biol Med. 1966 Dec;123(3):712-5. doi: 10.3181/00379727-123-31584.
10
The significance of inhibition of prostaglandin synthesis in the selection of non-steroidal anti-inflammatory agents.前列腺素合成抑制在非甾体抗炎药选择中的意义。
Int J Clin Pharmacol Biopharm. 1975 Jul;12(1-2):186-91.

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BitterDB database analysis plus cell stiffness screening identify flufenamic acid as the most potent TAS2R14-based relaxant of airway smooth muscle cells for therapeutic bronchodilation.苦味受体数据库分析联合细胞硬度筛选发现氟灭酸是气道平滑肌细胞中基于 TAS2R14 的最强松弛剂,可用于治疗性支气管扩张。
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From Low-Grade Inflammation in Osteoarthritis to Neuropsychiatric Sequelae: A Narrative Review.
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Oxicam-type non-steroidal anti-inflammatory drugs inhibit NPR1-mediated salicylic acid pathway.昔布类非甾体抗炎药抑制 NPR1 介导的水杨酸途径。
Nat Commun. 2021 Dec 15;12(1):7303. doi: 10.1038/s41467-021-27489-w.
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Case Report: Changes of Vascular Reactivity and Arterial Stiffness in a Patient With Covid-19 Infection.病例报告:一名新冠肺炎感染患者的血管反应性和动脉僵硬度变化
Front Cardiovasc Med. 2021 May 12;8:671669. doi: 10.3389/fcvm.2021.671669. eCollection 2021.
6
Fever During Localized Inflammation in Mice Is Elicited by a Humoral Pathway and Depends on Brain Endothelial Interleukin-1 and Interleukin-6 Signaling and Central EP Receptors.在小鼠的局部炎症中,发热是由体液途径引发的,并且依赖于脑内皮细胞白细胞介素-1 和白细胞介素-6 信号以及中枢 EP 受体。
J Neurosci. 2021 Jun 16;41(24):5206-5218. doi: 10.1523/JNEUROSCI.0313-21.2021. Epub 2021 May 3.
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Enhancing Anti-Tumor Activity of Sorafenib Mesoporous Silica Nanomatrix in Metastatic Breast Tumor and Hepatocellular Carcinoma via the Co-Administration with Flufenamic Acid.通过与氟芬那酸联合给药增强索拉非尼介孔硅纳米基质在转移性乳腺癌和肝癌中的抗肿瘤活性。
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Fenamates Inhibit Human Sodium Channel Nav1.2 and Protect Glutamate-Induced Injury in SH-SY5Y Cells.非甾体抗炎芬那酸类药物抑制人钠通道Nav1.2并保护SH-SY5Y细胞免受谷氨酸诱导的损伤。
Cell Mol Neurobiol. 2020 Nov;40(8):1405-1416. doi: 10.1007/s10571-020-00826-1. Epub 2020 Mar 11.
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Molecules. 2019 May 11;24(9):1820. doi: 10.3390/molecules24091820.
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Flufenamic acid inhibits secondary hemorrhage and BSCB disruption after spinal cord injury.氟灭酸抑制脊髓损伤后的二次出血和血脑屏障破坏。
Theranostics. 2018 Jul 30;8(15):4181-4198. doi: 10.7150/thno.25707. eCollection 2018.