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Alterations of cell surfaces as a pathogenetic factor in psoriasis. Possible loss of contact inhibition of growth.

作者信息

Orfanos C E, Schaumburg-Lever G, Mahrle G, Lever W F

出版信息

Arch Dermatol. 1973 Jan;107(1):38-46.

PMID:4566189
Abstract
摘要

相似文献

1
Alterations of cell surfaces as a pathogenetic factor in psoriasis. Possible loss of contact inhibition of growth.细胞表面改变作为银屑病的发病因素。生长接触抑制可能丧失。
Arch Dermatol. 1973 Jan;107(1):38-46.
2
Studies on the ultrastructure of psoriasis and of the "normal" skin of psoriatics.银屑病及银屑病患者“正常”皮肤的超微结构研究。
Acta Derm Venereol. 1971;51(6):435-43.
3
Metabolic and ultrastructural studies in a patient with pustular psoriasis (von Zumbusch).一名脓疱型银屑病（冯·祖姆布施型）患者的代谢及超微结构研究
Arch Dermatol. 1972 Feb;105(2):189-96.
4
[Psoriasis].[银屑病]
Rev Prat. 2006 Dec 15;56(19):2153-8.
5
Psoriasis: current concepts in management.银屑病：当前的管理理念
Drugs. 1977 Apr;13(4):288-96. doi: 10.2165/00003495-197713040-00004.
6
Aspects of the Odland bodies in the synthesis of keratohyalin.奥尔德体在透明角质颗粒合成中的作用
Acta Derm Venereol. 1974;54(5):359-64.
7
The action of antimetabolites in the skin: methotrexate for psoriasis.抗代谢物在皮肤中的作用：甲氨蝶呤治疗银屑病。
Adv Biol Skin. 1972;12:287-302.
8
Biochemical and pathophysiological rationale for amethopterin in psoriasis.氨甲蝶呤治疗银屑病的生化及病理生理原理
Ann N Y Acad Sci. 1971 Nov 30;186:452-66. doi: 10.1111/j.1749-6632.1971.tb47001.x.
9
Ultrastructural findings and tumor necrosis factor-alpha and intercellular adhesion molecule-1 expression in psoriasis patients before and after oral cyclosporin A therapy.口服环孢素A治疗前后银屑病患者的超微结构 findings 以及肿瘤坏死因子-α和细胞间黏附分子-1表达
Ultrastruct Pathol. 2006 Jan-Feb;30(1):95-102. doi: 10.1080/01913120500406616.
10
Psoriasis-liver-methotrexate interactions.银屑病-肝脏-甲氨蝶呤相互作用
Arch Dermatol. 1973 Jul;108(1):36-42.

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Collagenase expression in transgenic mouse skin causes hyperkeratosis and acanthosis and increases susceptibility to tumorigenesis.转基因小鼠皮肤中的胶原酶表达会导致角化过度和棘皮症，并增加肿瘤发生的易感性。
Mol Cell Biol. 1995 Oct;15(10):5732-9. doi: 10.1128/MCB.15.10.5732.
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Incorporation of 14C-linoleic acid in cerebrosides of psoriatic and normal human skin.14C-亚油酸在银屑病患者和正常人皮肤脑苷脂中的掺入情况。
Arch Dermatol Res. 1981;270(4):441-4. doi: 10.1007/BF00403788.
3
Domain turnover of junctional membrane areas in the epidermis.
表皮中连接膜区域的结构域更新。
Arch Dermatol Res. 1982;274(1-2):93-9. doi: 10.1007/BF00510363.
4
Immunofluorescence in psoriasis: studies of immunoglobulins, complement deposits, and three membrane markers.银屑病中的免疫荧光：免疫球蛋白、补体沉积及三种膜标志物的研究
Arch Dermatol Res. 1981;271(3):295-303. doi: 10.1007/BF00409458.
5
Autoradiographic study of glycoprotein synthesis during embryonic epidermogenesis of the mouse in vitro.小鼠胚胎表皮发生体外过程中糖蛋白合成的放射自显影研究。
Arch Dermatol Res. 1983;275(2):109-13. doi: 10.1007/BF00412885.
6
Scanning electron microscopy of skin surface and the internal structure of corneocyte in normal human skin. An application of the osmium-dimethyl sulfoxide-osmium method.
Arch Dermatol Res. 1988;280(5):293-9. doi: 10.1007/BF00440603.
7
Histopathogenesis of inflammatory linear verrucose epidermal naevus: histochemistry, immunohistochemistry and ultrastructure.炎症性线状疣状表皮痣的组织病理学发生机制：组织化学、免疫组织化学及超微结构
Arch Dermatol Res. 1991;283(8):491-9. doi: 10.1007/BF00371921.
8
Expression, topography, and function of integrin receptors are severely altered in keratinocytes from involved and uninvolved psoriatic skin.
J Clin Invest. 1992 Jun;89(6):1783-95. doi: 10.1172/JCI115782.
9
[Lack of beta-adrenergic stimulation of membrane bound adenyl cyclase in psoriasis as compared to normal epidermis (author's transl)].与正常表皮相比，银屑病中膜结合腺苷酸环化酶缺乏β-肾上腺素能刺激（作者译）
Arch Dermatol Res (1975). 1975 Sep 12;253(2):195-202. doi: 10.1007/BF00582071.
10
Long-term tissue culture of epithelial-like cells from human skin (NCTC strain 2544). I. Measurement of viscosity.人皮肤上皮样细胞的长期组织培养（NCTC 2544株）。I. 粘度测量
Arch Dermatol Res (1975). 1976 Oct 27;256(3):255-60. doi: 10.1007/BF00572490.