细胞表面改变作为银屑病的发病因素。生长接触抑制可能丧失。 - Suppr

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超能文献

Alterations of cell surfaces as a pathogenetic factor in psoriasis. Possible loss of contact inhibition of growth.

作者信息

Orfanos C E, Schaumburg-Lever G, Mahrle G, Lever W F

出版信息

Arch Dermatol. 1973 Jan;107(1):38-46.

PMID:4566189

Abstract

摘要

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Alterations of cell surfaces as a pathogenetic factor in psoriasis. Possible loss of contact inhibition of growth.细胞表面改变作为银屑病的发病因素。生长接触抑制可能丧失。

Arch Dermatol. 1973 Jan;107(1):38-46.

Studies on the ultrastructure of psoriasis and of the "normal" skin of psoriatics.银屑病及银屑病患者“正常”皮肤的超微结构研究。

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Psoriasis-liver-methotrexate interactions.银屑病-肝脏-甲氨蝶呤相互作用

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Autoradiographic study of glycoprotein synthesis during embryonic epidermogenesis of the mouse in vitro.小鼠胚胎表皮发生体外过程中糖蛋白合成的放射自显影研究。

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[Lack of beta-adrenergic stimulation of membrane bound adenyl cyclase in psoriasis as compared to normal epidermis (author's transl)].与正常表皮相比，银屑病中膜结合腺苷酸环化酶缺乏β-肾上腺素能刺激（作者译）

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Long-term tissue culture of epithelial-like cells from human skin (NCTC strain 2544). I. Measurement of viscosity.人皮肤上皮样细胞的长期组织培养（NCTC 2544株）。I. 粘度测量

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