Khitrov N K, Svistukhin A I, Tezikov E B
Biull Eksp Biol Med. 1979 Jun;87(6):523-5.
Preliminary sympathectomia depletes acetylcholine (ACh) in the heart of rabbits under hypoxia. In these conditions the inhibitory action of ACh on the rat isolated heart is reduced under the noradrenaline content fall, while under increase it is potentiated. Under hypoxia noradrenaline increases concentration of potassium in the myocardium, thus stimulating ACh formation and activity. It is suggested that under deep hypoxia suppression of the sympathetic mechanisms causes functional isolation of the heart from nervous effects.
初步的交感神经切除术会使处于缺氧状态的兔子心脏中的乙酰胆碱（ACh）耗竭。在这些情况下，当去甲肾上腺素含量下降时，ACh对大鼠离体心脏的抑制作用减弱，而当去甲肾上腺素含量增加时，该抑制作用增强。在缺氧状态下，去甲肾上腺素会增加心肌中的钾浓度，从而刺激ACh的形成和活性。有人认为，在深度缺氧状态下，交感神经机制的抑制会导致心脏与神经效应发生功能隔离。
