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Capsaicin-evoked release of substance P from primary sensory neurons.

作者信息

Theriault E, Otsuka M, Jessell T

出版信息

Brain Res. 1979 Jul 6;170(1):209-13. doi: 10.1016/0006-8993(79)90957-0.

DOI:10.1016/0006-8993(79)90957-0
PMID:466404
Abstract
摘要

相似文献

1
Capsaicin-evoked release of substance P from primary sensory neurons.
Brain Res. 1979 Jul 6;170(1):209-13. doi: 10.1016/0006-8993(79)90957-0.
2
Thermal analgesia and substance P depletion induced by capsaicin in guinea-pigs.辣椒素在豚鼠中诱导的热镇痛和P物质耗竭
Neuroscience. 1981;6(11):2217-22. doi: 10.1016/0306-4522(81)90009-9.
3
Effects of capsaicin on nociceptive heat, pressure and chemical thresholds and on substance P levels in the rat.辣椒素对大鼠伤害性热、压力和化学阈值以及P物质水平的影响。
Brain Res. 1980 May 12;189(2):561-4. doi: 10.1016/0006-8993(80)90369-8.
4
Intrathecal capsaicin depletes substance P in the rat spinal cord and produces prolonged thermal analgesia.
Science. 1979 Oct 26;206(4417):481-3. doi: 10.1126/science.228392.
5
Effect of capsaicin pretreatment on capsaicin-evoked release of immunoreactive somatostatin and substance P from primary sensory neurons.辣椒素预处理对辣椒素诱发的初级感觉神经元中免疫反应性生长抑素和P物质释放的影响。
Naunyn Schmiedebergs Arch Pharmacol. 1981 Feb;316(1):38-41. doi: 10.1007/BF00507224.
6
Substance P release from spinal cord slices by capsaicin.辣椒素诱导脊髓切片释放P物质。
Life Sci. 1979 Aug 13;25(7):629-36. doi: 10.1016/0024-3205(79)90558-7.
7
Capsaicinoid-induced local and systemic antinociception without substance P depletion.辣椒素诱导的局部和全身镇痛作用,而不伴有P物质耗竭。
Brain Res. 1982 Jul 22;244(1):193-7. doi: 10.1016/0006-8993(82)90921-0.
8
Effect of capsaicin administration to neonatal rats on the substance P content of discrete CNS regions.
Brain Res. 1981 Oct 19;222(2):428-31. doi: 10.1016/0006-8993(81)91049-0.
9
Intraventricular capsaicin: alterations in analgesic responsivity without depletion of substance P.脑室内注射辣椒素:镇痛反应性改变而P物质未耗竭
Neuroscience. 1982 Mar;7(3):631-8. doi: 10.1016/0306-4522(82)90068-9.
10
Relationship between capsaicin-evoked substance P release and neurokinin 1 receptor internalization in the rat spinal cord.辣椒素诱发的P物质释放与大鼠脊髓中神经激肽1受体内化之间的关系。
Neuroscience. 2003;118(2):535-45. doi: 10.1016/s0306-4522(02)00977-6.

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蛋白水解作用控制内源性 P 物质水平。
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Substance P-driven feed-forward inhibitory activity in the mammalian spinal cord.P物质驱动的哺乳动物脊髓前馈抑制活性。
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The effect of cetirizine and loratadine on codeine-induced histamine release in human skin in vivo assessed by cutaneous microdialysis.通过皮肤微透析评估西替利嗪和氯雷他定对体内人皮肤中可待因诱导的组胺释放的影响。
Inflamm Res. 1996 Sep;45(9):486-90. doi: 10.1007/BF02252321.
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Selective phototoxic destruction of rat Merkel cells abolishes responses of slowly adapting type I mechanoreceptor units.对大鼠默克尔细胞进行选择性光毒性破坏可消除慢适应性I型机械感受器单位的反应。
J Physiol. 1994 Sep 1;479 ( Pt 2)(Pt 2):247-56. doi: 10.1113/jphysiol.1994.sp020292.
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Muscarinic excitatory and inhibitory mechanisms involved in afferent fibre-evoked depolarization of motoneurones in the neonatal rat spinal cord.新生大鼠脊髓中运动神经元传入纤维诱发去极化所涉及的毒蕈碱兴奋和抑制机制。
Br J Pharmacol. 1993 Sep;110(1):61-70. doi: 10.1111/j.1476-5381.1993.tb13772.x.
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Supersensitivity of the rabbit iris sphincter muscle induced by trigeminal denervation: the role of substance P.三叉神经去神经支配诱导兔虹膜括约肌超敏反应:P物质的作用
J Physiol. 1984 May;350:583-97. doi: 10.1113/jphysiol.1984.sp015219.
10
Capsaicin causes release of a substance P-like peptide in guinea-pig inferior mesenteric ganglia.辣椒素可使豚鼠肠系膜下神经节释放一种P物质样肽。
J Physiol. 1983 Jul;340:107-20. doi: 10.1113/jphysiol.1983.sp014752.