Bonmassar A
Experientia. 1979 Aug 15;35(8):1098-9. doi: 10.1007/BF01949965.
Congenic mice were sensitized with viable H-2-incompatible radiation-induced lymphomas (RIL), challenged with syngeneic RIL and treated with bis-chloroethyl-nitrosourea. Either enhancement or inhibition of RIL was found in presensitized mice, depending on the tumor-host system used.
将同基因小鼠用活的H-2不相容辐射诱导淋巴瘤(RIL)致敏,用同基因RIL攻击并用双氯乙基亚硝脲治疗。在预先致敏的小鼠中,根据所使用的肿瘤-宿主系统,发现RIL要么增强要么受到抑制。