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Enlargement of epididymal adipocytes in relation to hyperinsulinemia in obese hyperglycemic mice (ob-ob).

作者信息

Joosten H F, van der Kroon P H

出版信息

Metabolism. 1974 Jan;23(1):59-66. doi: 10.1016/0026-0495(74)90104-8.

DOI:10.1016/0026-0495(74)90104-8
PMID:4808512
Abstract
摘要

相似文献

1
Enlargement of epididymal adipocytes in relation to hyperinsulinemia in obese hyperglycemic mice (ob-ob).肥胖高血糖小鼠(ob-ob)附睾脂肪细胞增大与高胰岛素血症的关系。
Metabolism. 1974 Jan;23(1):59-66. doi: 10.1016/0026-0495(74)90104-8.
2
Selective inhibition of 11 beta-hydroxysteroid dehydrogenase type 1 improves hepatic insulin sensitivity in hyperglycemic mice strains.选择性抑制11β-羟类固醇脱氢酶1型可改善高血糖小鼠品系的肝脏胰岛素敏感性。
Endocrinology. 2003 Nov;144(11):4755-62. doi: 10.1210/en.2003-0344. Epub 2003 Jul 31.
3
Insulin receptor deficiency in genetic and acquired obesity.遗传和后天肥胖中的胰岛素受体缺乏症。
J Clin Invest. 1975 Oct;56(4):769-80. doi: 10.1172/JCI108155.
4
Myeloid-derived miR-6236 potentiates adipocyte insulin signaling and prevents hyperglycemia during obesity.髓系衍生的 miR-6236 增强脂肪细胞胰岛素信号传导,预防肥胖期间的高血糖。
Nat Commun. 2024 Jun 25;15(1):5394. doi: 10.1038/s41467-024-49632-z.
5
Regulation of resistin expression and circulating levels in obesity, diabetes, and fasting.肥胖、糖尿病和禁食状态下抵抗素表达及循环水平的调控
Diabetes. 2004 Jul;53(7):1671-9. doi: 10.2337/diabetes.53.7.1671.
6
[Conditioning of hyperglycemia and hyperinsulinemia in genetically obese mice through resistance to insulin].通过对胰岛素的抵抗来调节遗传性肥胖小鼠的高血糖和高胰岛素血症
Fiziol Zh. 1974 Jan-Feb;20(2):265-72.
7
Glucose tolerance and time sequence of adiposity, hyperinsulinemia and hyperglycemia in obese-hyperglycemic mice (obob).肥胖高血糖小鼠(obob)的糖耐量以及肥胖、高胰岛素血症和高血糖的时间顺序
Metabolism. 1970 Sep;19(9):687-93. doi: 10.1016/0026-0495(70)90065-x.
8
The development of obesity, hyperinsulinemia, and hyperglycemia in ob/ob mice.ob/ob小鼠肥胖、高胰岛素血症和高血糖的发展过程。
Metabolism. 1976 Dec;25(12):1567-74. doi: 10.1016/0026-0495(76)90109-8.
9
Degradation of insulin in vitro by liver and epididymal adipose tissue from obese-hyperglycaemic mice.肥胖高血糖小鼠肝脏和附睾脂肪组织对胰岛素的体外降解作用。
Biochem J. 1968 Jan;106(2):543-7. doi: 10.1042/bj1060543.
10
Lipolysis of genetically obese and-or hyperglycemic mice with reference to insulin response of adipose tissue.参照脂肪组织的胰岛素反应对遗传性肥胖和/或高血糖小鼠进行脂肪分解研究。
Horm Metab Res. 1972 Sep;4(5):331-7. doi: 10.1055/s-0028-1094027.

引用本文的文献

1
WNIN/GR-Ob - an insulin-resistant obese rat model from inbred WNIN strain.WNIN/GR-Ob-一种来自近交系 WNIN 品系的胰岛素抵抗肥胖大鼠模型。
Indian J Med Res. 2011 Sep;134(3):320-9.
2
Melanin-concentrating hormone overexpression in transgenic mice leads to obesity and insulin resistance.转基因小鼠中黑色素浓集激素的过度表达会导致肥胖和胰岛素抵抗。
J Clin Invest. 2001 Feb;107(3):379-86. doi: 10.1172/JCI10660.
3
Age-related changes in adenohypophyses of mice with the hereditary obese hyperglycemic syndrome (ob/ob) in relation to the diabetic state of the animals.
遗传性肥胖高血糖综合征(ob/ob)小鼠腺垂体的年龄相关变化与动物糖尿病状态的关系。
Acta Diabetol Lat. 1982 Jul-Sep;19(3):227-32. doi: 10.1007/BF02624682.
4
Abnormal plasma glucose and insulin responses in heterozygous lean (ob/+) mice.杂合子瘦型(ob/+)小鼠的血浆葡萄糖和胰岛素反应异常。
Diabetologia. 1981 May;20(5):573-7. doi: 10.1007/BF00252768.
5
Hyperinsulinemia in pre-weaning diabetes (db) mice.
Diabetologia. 1974 Nov;10 Suppl:607-10. doi: 10.1007/BF01221993.
6
Effect of high fat vs. high carbohydrate feeding on the development of obesity in weanling ob/ob mice.
Diabetologia. 1976 May;12(2):155-9. doi: 10.1007/BF00428982.
7
Obesity: do fat cells from genetically obese mice (C57BL/6J ob/ob) have an innate capacity for increased fat storage?肥胖:来自遗传性肥胖小鼠(C57BL/6J ob/ob)的脂肪细胞是否具有增加脂肪储存的先天能力?
Diabetologia. 1978 Dec;15(6):465-70. doi: 10.1007/BF02342871.
8
Obese and diabetes: two mutant genes causing diabetes-obesity syndromes in mice.肥胖与糖尿病:导致小鼠出现糖尿病 - 肥胖综合征的两个突变基因。
Diabetologia. 1978 Mar;14(3):141-8. doi: 10.1007/BF00429772.
9
Lipolysis and cyclic AMP levels in epididymal adipose tissue of obese-hyperglycaemic mice.肥胖高血糖小鼠附睾脂肪组织中的脂肪分解与环磷酸腺苷水平
Diabetologia. 1977 Dec;13(6):553-61. doi: 10.1007/BF01236307.
10
An enzymatic defect in the obese (ob/ob) mouse: loss of thyroid-induced sodium- and potassium-dependent adenosinetriphosphatase.肥胖(ob/ob)小鼠的一种酶缺陷:甲状腺诱导的钠钾依赖型三磷酸腺苷酶缺失。
Proc Natl Acad Sci U S A. 1978 Jan;75(1):477-81. doi: 10.1073/pnas.75.1.477.