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Cell cycle dependence of chemically induced malignant transformation in vitro.

作者信息

Marquardt H

出版信息

Cancer Res. 1974 Jul;34(7):1612-5.

PMID:4857730
Abstract
摘要

相似文献

1
Cell cycle dependence of chemically induced malignant transformation in vitro.体外化学诱导恶性转化的细胞周期依赖性
Cancer Res. 1974 Jul;34(7):1612-5.
2
Proceedings: Cell cycle dependence of chemically induced malignant transformation in vitro.论文集：体外化学诱导恶性转化的细胞周期依赖性
Naunyn Schmiedebergs Arch Pharmacol. 1974;282(Suppl):suppl 282:R63.
3
Quantitative and qualitative studies of chemical transformation of cloned C3H mouse embryo cells sensitive to postconfluence inhibition of cell division.对汇合后细胞分裂抑制敏感的克隆C3H小鼠胚胎细胞化学转化的定量和定性研究。
Cancer Res. 1973 Dec;33(12):3239-49.
4
Quantitation of chemically induced neoplastic transformation of BALB-3T3 cloned cell lines.化学诱导的BALB - 3T3克隆细胞系肿瘤转化的定量分析。
Cancer Res. 1972 Dec;32(12):2686-95.
5
Cell cycle dependency of DNA damage and repair in transformable mouse fibroblasts treated with N-methyl-N'--nitro-N-nitrosoguanidine.用N-甲基-N'-硝基-N-亚硝基胍处理的可转化小鼠成纤维细胞中DNA损伤与修复的细胞周期依赖性
Cancer Res. 1974 Jul;34(7):1600-7.
6
Loss of density-dependent regulation of multiplication of BALB-3T3 cells chemically transformed in vitro.体外化学转化的BALB-3T3细胞增殖的密度依赖性调控丧失。
J Cell Physiol. 1973 Feb;81(1):133-8. doi: 10.1002/jcp.1040810116.
7
Cell cycle dependency of oncogenic transformation induced by N-methyl-N'-nitro-N-nitrosoquanidine in culture.培养条件下N-甲基-N'-硝基-N-亚硝基胍诱导致癌转化的细胞周期依赖性
Cancer Res. 1974 Mar;34(3):526-37.
8
Changes in the uptake of 2-deoxy-D-glucose in BALB-3T3 cells chemically transformed in culture.
J Cell Physiol. 1974 Apr;83(2):193-201. doi: 10.1002/jcp.1040830205.
9
Malignant transformation in vitro of mouse fibroblasts by 7,12-dimethylbenz(A)anthracene and 7-hydroxymethylbenz(A)anthracene and by their K-region derivatives.7,12-二甲基苯并(a)蒽和7-羟甲基苯并(a)蒽及其K区域衍生物对小鼠成纤维细胞的体外恶性转化作用。
Int J Cancer. 1974 Mar 15;13(3):304-10. doi: 10.1002/ijc.2910130305.
10
Polyriboinosinic-polyribocytidylic acid prevents chemically-induced malignant transformation in vitro.
Nat New Biol. 1973 Dec 19;246(155):228-9. doi: 10.1038/newbio246228a0.

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Mechanisms of DNA-reactive and epigenetic chemical carcinogens: applications to carcinogenicity testing and risk assessment.DNA反应性和表观遗传化学致癌物的作用机制：在致癌性测试和风险评估中的应用
Toxicol Res (Camb). 2018 Dec 18;8(2):123-145. doi: 10.1039/c8tx00250a. eCollection 2019 Mar 1.
2
Differential cell cycle phase specificity for neoplastic transformation and mutation to ouabain resistance induced by N-methyl-N'-nitro-N-nitrosoguanidine in synchronized C3H10T 1/2 C18 cells.在同步化的C3H10T 1/2 C18细胞中，N-甲基-N'-硝基-N-亚硝基胍诱导肿瘤转化及对哇巴因抗性突变的细胞周期阶段特异性差异
Proc Natl Acad Sci U S A. 1982 Jul;79(14):4342-6. doi: 10.1073/pnas.79.14.4342.
3
DNA precursor pool: a significant target for N-methyl-N-nitrosourea in C3H/10T1/2 clone 8 cells.
DNA前体池：N-甲基-N-亚硝基脲在C3H/10T1/2克隆8细胞中的一个重要靶点。
Proc Natl Acad Sci U S A. 1982 Apr;79(7):2211-5. doi: 10.1073/pnas.79.7.2211.
4
Cell cycle-specific mutagenesis at the hypoxanthine phosphoribosyltransferase locus in adult rat liver epithelial cells.成年大鼠肝上皮细胞中次黄嘌呤磷酸核糖基转移酶基因座的细胞周期特异性诱变
Proc Natl Acad Sci U S A. 1980 Dec;77(12):7377-9. doi: 10.1073/pnas.77.12.7377.
5
Cycle-related toxicity and transformation in 10T1/2 cells treated with N-methyl-N'-nitro-N-nitrosoguanidine.用N-甲基-N'-硝基-N-亚硝基胍处理的10T1/2细胞中的周期相关毒性和转化
Proc Natl Acad Sci U S A. 1980 Aug;77(8):4813-7. doi: 10.1073/pnas.77.8.4813.
6
In vitro approaches for determining mechanisms of toxicity and carcinogenicity by asbestos in the gastrointestinal and respiratory tracts.通过体外方法确定石棉在胃肠道和呼吸道中的毒性及致癌机制。
Environ Health Perspect. 1983 Nov;53:155-61. doi: 10.1289/ehp.8353155.
7
Interaction of asbestos with metaplastic squamous epithelium developing in organ cultures of hamster trachea.石棉与仓鼠气管器官培养中发育的化生鳞状上皮的相互作用。
Environ Health Perspect. 1983 Sep;51:27-33. doi: 10.1289/ehp.835127.
8
Differentiation of the mammary gland and susceptibility to carcinogenesis.乳腺的分化与致癌易感性。
Breast Cancer Res Treat. 1982;2(1):5-73. doi: 10.1007/BF01805718.
9
DNA adducts and cell cycle.DNA加合物与细胞周期
J Cancer Res Clin Oncol. 1986;112(3):189-95. doi: 10.1007/BF00395911.
10
Recovery patterns of rat hemopoietic stem cells between pulse doses of 7,12-dimethylbenz(a)anthracene (DMBA) applied in a leukemogenic regimen.在致白血病方案中应用的7,12-二甲基苯并(a)蒽（DMBA）脉冲剂量之间大鼠造血干细胞的恢复模式。
J Cancer Res Clin Oncol. 1986;111(3):237-42. doi: 10.1007/BF00389239.