Tyler B, Magasanik B
J Bacteriol. 1969 Feb;97(2):550-6. doi: 10.1128/jb.97.2.550-556.1969.
The molecular basis of transient repression of beta-galactosidase by glucose was examined. This repression acted only at the level of transcription. Apparently, it was not mediated by the I-gene product. Analysis of single cells in a culture subjected to transient repression showed that essentially all cells initially experienced repression and later became gradually resistant to repression.
研究了葡萄糖对β-半乳糖苷酶瞬时抑制的分子基础。这种抑制仅作用于转录水平。显然,它不是由I基因产物介导的。对处于瞬时抑制状态的培养物中的单细胞进行分析表明,基本上所有细胞最初都经历了抑制,随后逐渐对抑制产生抗性。