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Depressed myocardial creatine phosphokinase activity following experimental myocardial infarction in rabbit.

作者信息

Kjekshus J K, Sobel B E

出版信息

Circ Res. 1970 Sep;27(3):403-14. doi: 10.1161/01.res.27.3.403.

DOI:10.1161/01.res.27.3.403
PMID:5452738
Abstract
摘要

相似文献

1
Depressed myocardial creatine phosphokinase activity following experimental myocardial infarction in rabbit.兔实验性心肌梗死后心肌肌酸磷酸激酶活性降低
Circ Res. 1970 Sep;27(3):403-14. doi: 10.1161/01.res.27.3.403.
2
[Creatine phosphokinase and ATPase activity of the extrainfarct areas of the myocardium in rabbits after a coronary-occluding infarct with alpha-tocopherol and sodium nucleinate administration].[给予α-生育酚和核酸钠后，兔冠状动脉闭塞性梗死心肌梗死灶外区域的肌酸磷酸激酶和ATP酶活性]
Patol Fiziol Eksp Ter. 1986 Jan-Feb(1):51-3.
3
[Studies on myocardial infarct size and the release of myocardial enzymes in experimental infarction: correlation of infarct size and released myocardial enzymes after coronary artery ligation and reperfusion with special reference to CPK-MB].[实验性心肌梗死中心肌梗死面积及心肌酶释放的研究：冠状动脉结扎及再灌注后梗死面积与释放的心肌酶的相关性，特别提及肌酸磷酸激酶同工酶MB]
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Canine myocardial creatine kinase isoenzyme response to coronary artery occlusion.犬心肌肌酸激酶同工酶对冠状动脉阻塞的反应。
Am J Physiol. 1989 Feb;256(2 Pt 2):H508-14. doi: 10.1152/ajpheart.1989.256.2.H508.
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Quantitative assessment of the extent of myocardial infarction in the conscious dog by means of analysis of serial changes in serum creatine phosphokinase activity.通过分析血清肌酸磷酸激酶活性的系列变化对清醒犬心肌梗死范围进行定量评估。
J Clin Invest. 1971 Dec;50(12):2614-25. doi: 10.1172/JCI106762.
6
Molecular changes in myocardial infarction in heart muscle.心肌梗死时心肌的分子变化。
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7
Absence of a lateral border zone of intermediate creatine phosphokinase depletion surrounding a central infarct 24 hours after acute coronary occlusion in the dog.犬急性冠状动脉闭塞24小时后，梗死灶中心周围未出现中间型肌酸磷酸激酶耗竭的外侧边界区。
Circ Res. 1977 Nov;41(5):673-83. doi: 10.1161/01.res.41.5.673.
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On the creatine phosphokinase of heart muscle mitochondria.关于心肌线粒体的肌酸磷酸激酶
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Accumulation of gallium-67 in regions of acute myocardial infarction.镓-67在急性心肌梗死区域的蓄积。
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Demonstration of creatine phosphokinase in human myocardial slices.人体心肌切片中肌酸磷酸激酶的显示
J Clin Pathol. 1979 Feb;32(2):196-7. doi: 10.1136/jcp.32.2.196.

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Deleterious effects of lack of cardiac PAI-1 after coronary occlusion in mice and their pathophysiologic determinants.小鼠冠状动脉闭塞后心脏PAI - 1缺乏的有害影响及其病理生理决定因素。
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Cardioprotective effect of insulin-like growth factor I in myocardial ischemia followed by reperfusion.胰岛素样生长因子I在心肌缺血再灌注中的心脏保护作用。
Proc Natl Acad Sci U S A. 1995 Aug 15;92(17):8031-5. doi: 10.1073/pnas.92.17.8031.
3
Experimental myocardial infarction in rats acclimated to simulated high altitude.
Basic Res Cardiol. 1980 Jul-Aug;75(4):544-54. doi: 10.1007/BF01907836.
4
Comparison of enzymic with cineangiocardiographic estimations of myocardial infarct size.心肌梗死面积的酶学测定与心血管造影测定的比较。
Br Heart J. 1980 Jun;43(6):609-16. doi: 10.1136/hrt.43.6.609.
5
Mechanism of the beneficial effect of dexamethasone on myocardial cell integrity in acure myocardial ischemia.地塞米松对急性心肌缺血中心肌细胞完整性有益作用的机制
Basic Res Cardiol. 1980 Mar-Apr;75(2):328-39. doi: 10.1007/BF01907581.
6
Deleterious influence of hypothyroidism on evolving myocardial infarction in conscious dogs.
J Clin Invest. 1981 Apr;67(4):1024-34. doi: 10.1172/jci110113.
7
The inability of isoproterenol or propranolol to alter the lateral dimensions of experimentally induced myocardial infarcts.异丙肾上腺素或普萘洛尔无法改变实验性诱导心肌梗死的横向尺寸。
Basic Res Cardiol. 1982 Sep-Oct;77(5):486-98. doi: 10.1007/BF01907941.
8
Limitation of myocardial infarct size in patients less than 66 years treated with alprenolol.阿普洛尔治疗66岁以下患者时对心肌梗死面积的限制作用。
Br Heart J. 1981 May;45(5):583-8. doi: 10.1136/hrt.45.5.583.
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The Szasz-ratio (CK/GOT) as example for the diagnostic significance of enzyme ratios in serum.以萨斯比率（肌酸激酶/谷草转氨酶）为例说明血清中酶比率的诊断意义。
Klin Wochenschr. 1980 Jul 15;58(14):709-18. doi: 10.1007/BF01478458.
10
Production of cardiac lesions with tyramine in intact rats. Studies on serum and myocardial creatine kinase activity changes and ultrastructural aspects.完整大鼠中酪胺诱导心脏病变的研究。血清和心肌肌酸激酶活性变化及超微结构方面的研究。
Basic Res Cardiol. 1983 May-Jun;78(3):289-97. doi: 10.1007/BF01907438.