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本文引用的文献

1
The influence of certain salts, amino acids, sugars, and proteins on the stability of rickettsiae.某些盐类、氨基酸、糖类及蛋白质对立克次氏体稳定性的影响。
J Bacteriol. 1950 Apr;59(4):509-22. doi: 10.1128/jb.59.4.509-522.1950.
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Histocompatibility-linked genetic control of disease susceptibility. Murine lymphocytic choriomeningitis virus infection.疾病易感性的组织相容性连锁基因控制。小鼠淋巴细胞性脉络丛脑膜炎病毒感染。
J Exp Med. 1973 May 1;137(5):1201-12. doi: 10.1084/jem.137.5.1201.
3
The pathogenesis of respiratory syncytial virus infection in infant ferrets.婴儿雪貂呼吸道合胞病毒感染的发病机制。
Am J Pathol. 1976 Feb;82(2):339-52.
4
X-linked B-lymphocyte defect in CBA/N mice. III. Abnormal development of B-lymphocyte populations defined by their density of surface immunoglobulin.CBA/N小鼠的X连锁B淋巴细胞缺陷。III. 由表面免疫球蛋白密度定义的B淋巴细胞群体的异常发育。
J Exp Med. 1976 Aug 1;144(2):507-18. doi: 10.1084/jem.144.2.507.
5
Experimental respiratory syncytial virus infection of four species of primates.四种灵长类动物的实验性呼吸道合胞病毒感染
J Med Virol. 1977;1(3):157-62. doi: 10.1002/jmv.1890010302.
6
The pathogenesis of respiratory syncytial virus infection in cotton rats.棉鼠呼吸道合胞病毒感染的发病机制。
Am J Pathol. 1978 Dec;93(3):771-91.
7
Experimental respiratory syncytial virus pneumonia in cebus monkeys.卷尾猴实验性呼吸道合胞病毒肺炎
J Med Virol. 1978;2(1):45-59. doi: 10.1002/jmv.1890020108.
8
Evaluation of five temperature-sensitive mutants of respiratory syncytial virus in primates: I. Viral shedding, immunologic response, and associated illness.呼吸道合胞病毒五个温度敏感突变株在灵长类动物中的评估:I. 病毒排出、免疫反应及相关疾病
J Med Virol. 1978;3(2):91-100. doi: 10.1002/jmv.1890030202.

近交系小鼠中的呼吸道合胞病毒感染

Respiratory syncytial virus infection in inbred mice.

作者信息

Prince G A, Horswood R L, Berndt J, Suffin S C, Chanock R M

出版信息

Infect Immun. 1979 Nov;26(2):764-6. doi: 10.1128/iai.26.2.764-766.1979.

DOI:10.1128/iai.26.2.764-766.1979
PMID:546793
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC414679/
Abstract

Respiratory syncytial virus infected the nose and lungs of each of 20 strains of inbred mice, with viral titers varying 100-fold from least permissive to most permissive strains. Viral titers appeared to be under genetic control, but did not correlate with the H-2 haplotype.

摘要

呼吸道合胞病毒感染了20种近交系小鼠的鼻腔和肺部,病毒滴度在最不易感品系到最易感品系之间相差100倍。病毒滴度似乎受基因控制,但与H-2单倍型无关。