Kodama N, Mineura K, Suzuki J
No Shinkei Geka. 1977 Jan;5(1):65-9.
Two adult cases of cerebrovascular Moyamoya disease, associated with basilar aneurysm are reported. At the follow-up study of angiogram performed 14 months later in one case, the aneurysmal shadow was slightly larger than before. In the other case, craniotomy was performed and two aneurysms were confirmed to be the saccular aneurysms arising from the basilar head and superior cerebellar artery junction, respectively. From these facts, it is suggested that these aneurysms were not pseudo-aneurysms but sacuular aneurysms. As for the pseudo-aneurysm associated with cerebrovascular Moyamoya disease, we discussed in our previous report. It has been generally through that the mechanism of the occurence of symptoms in adult patient with cerebrovascular Moyamoya disease is due to rupture of abnormal vessels in the subarachnoid space. But from our experiences, it is suggested that there should be another mechanicsm, which begins with rupture of these basilar aneurysms. In addition to that, the growing mechanisms of basilar aneurysm associated with cerebrovascular Moyamoya disease is discussed.
报告了2例与基底动脉瘤相关的成人脑血管烟雾病病例。在其中1例患者14个月后进行的血管造影随访研究中，动脉瘤阴影比之前略大。在另一例患者中，进行了开颅手术，证实两个动脉瘤分别是起源于基底动脉头部和小脑上动脉交界处的囊状动脉瘤。基于这些事实，提示这些动脉瘤并非假性动脉瘤，而是囊状动脉瘤。关于与脑血管烟雾病相关的假性动脉瘤，我们在之前的报告中进行了讨论。一般认为，成年脑血管烟雾病患者出现症状的机制是蛛网膜下腔异常血管破裂。但根据我们的经验，提示可能存在另一种机制，即始于这些基底动脉瘤的破裂。此外，还讨论了与脑血管烟雾病相关的基底动脉瘤的生长机制。
