Meerson F Z, Kagan V E, Golubeva L Iu, Ugolev A A, Shimkovich M V
Kardiologiia. 1979 Aug;19(8):108-11.
It was shown in experiments on male Wistar rats that activation of peroxidized oxidation of myocardial lipids and accumulation of hydroperoxides in the myocardium comprise the decisive link of the pathogenic chain of stress and hypoxic damages to the heart. Injection of a potent non-toxic antioxidant into the animals before exposure to the stress or hypoxia blocks the activation of peroxidized oxidation and prevents disorder of the contractile function and damage to the heart muscle appraised according to the elimination of enzymes from the myocardial cells.
在对雄性Wistar大鼠进行的实验中表明，心肌脂质过氧化氧化的激活以及心肌中氢过氧化物的积累是应激和缺氧对心脏造成损害的致病链中的决定性环节。在动物暴露于应激或缺氧之前向其注射一种强效无毒抗氧化剂，可阻断过氧化氧化的激活，并防止根据心肌细胞中酶的消除情况评估的收缩功能紊乱和心肌损伤。
