De Renzi E, Faglioni P, Previdi P
Cortex. 1977 Dec;13(4):424-33. doi: 10.1016/s0010-9452(77)80022-1.
Corsi's cube test was given to 40 control and 80 brain-damaged patients to assess the relation of different aspects of spatial memory to the hemispheric locus of lesion. Spatial span was found affected by injury producing visual field defect (VFD), regardless of the side of the lesion. Delayed reproduction of a 3 cube sequence (which was within the span of every patient) was performed more poorly by patients with right hemisphere damage and VFD than by controls. This was true whether the delay was unfilled or filled with a counting activity, the two conditions being equally effective in bringing about the inferiority of the right brain-damaged group. Learning to criterion up to a maximum of 50 trails a supraspan sequence was failed by 65% of right brain-damaged patients with VFD, a percentage significantly higher than that found not only in the control group, but also in any other brain-damaged group. These findings point to the dominant role played by the posterior region of the right hemisphere in subserving spatial memory mechanisms, especially when the acquisition of stable traces is requested.
对40名对照组患者和80名脑损伤患者进行了科尔西方块测试,以评估空间记忆的不同方面与损伤半球位置之间的关系。发现空间广度受导致视野缺损(VFD)的损伤影响,而与损伤的部位无关。与对照组相比,右半球损伤且有视野缺损的患者对3个方块序列(每个患者都能完成该序列)的延迟再现表现更差。无论延迟期间是无填充活动还是进行计数活动,这两种情况在导致右脑损伤组表现较差方面同样有效。65%有视野缺损的右脑损伤患者在学习到最多50次试验的超广度序列时未能达到标准,这一百分比不仅显著高于对照组,也高于任何其他脑损伤组。这些发现表明,右半球后部区域在支持空间记忆机制方面起主导作用,尤其是在需要获取稳定痕迹时。