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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

J Physiol. 1967 Jul;191(2):309-24. doi: 10.1113/jphysiol.1967.sp008252.

During the initial stages of progressive hypoxia the intact, unanaesthetized chicken shows increases in heart rate and respiratory frequency with no change in arterial blood pressure and oxygen consumption. During the later stages, heart rate, diastolic and mean blood pressure and oxygen consumption fall, while respiratory frequency increases further.2. Following bilateral cervical vagotomy and adrenergic beta-receptor blockage there is no tachycardia, but the late bradycardia and fall in blood pressure do occur during progressive hypoxia. Respiratory frequency remains at a low level after vagotomy.3. It is suggested that the initial tachycardia is dependent on both the sympathetic and parasympathetic nervous systems, and that the former helps maintain arterial pressure during the early stages of hypoxia. Bradycardia and hypotension seem to be due to anoxia itself, and the vagus is essential for the increase in respiratory frequency.

在进行性缺氧的初始阶段，完整、未麻醉的鸡心率和呼吸频率增加，动脉血压和氧耗无变化。在后期，心率、舒张压和平均血压以及氧耗下降，而呼吸频率进一步增加。
双侧颈迷走神经切断术和肾上腺素能β受体阻断后，在进行性缺氧期间不会出现心动过速，但会出现后期心动过缓和血压下降。迷走神经切断术后呼吸频率维持在较低水平。
提示初始心动过速依赖于交感神经系统和副交感神经系统两者，并且前者在缺氧早期有助于维持动脉血压。心动过缓和低血压似乎是由于缺氧本身所致，并且迷走神经对于呼吸频率的增加至关重要。

J Physiol. 1967 Jul;191(2):309-24. doi: 10.1113/jphysiol.1967.sp008252.

During the initial stages of progressive hypoxia the intact, unanaesthetized chicken shows increases in heart rate and respiratory frequency with no change in arterial blood pressure and oxygen consumption. During the later stages, heart rate, diastolic and mean blood pressure and oxygen consumption fall, while respiratory frequency increases further.2. Following bilateral cervical vagotomy and adrenergic beta-receptor blockage there is no tachycardia, but the late bradycardia and fall in blood pressure do occur during progressive hypoxia. Respiratory frequency remains at a low level after vagotomy.3. It is suggested that the initial tachycardia is dependent on both the sympathetic and parasympathetic nervous systems, and that the former helps maintain arterial pressure during the early stages of hypoxia. Bradycardia and hypotension seem to be due to anoxia itself, and the vagus is essential for the increase in respiratory frequency.

在进行性缺氧的初始阶段，完整、未麻醉的鸡心率和呼吸频率增加，动脉血压和氧耗无变化。在后期，心率、舒张压和平均血压以及氧耗下降，而呼吸频率进一步增加。
双侧颈迷走神经切断术和肾上腺素能β受体阻断后，在进行性缺氧期间不会出现心动过速，但会出现后期心动过缓和血压下降。迷走神经切断术后呼吸频率维持在较低水平。
提示初始心动过速依赖于交感神经系统和副交感神经系统两者，并且前者在缺氧早期有助于维持动脉血压。心动过缓和低血压似乎是由于缺氧本身所致，并且迷走神经对于呼吸频率的增加至关重要。