Lodato G, Brancato G
J Fr Ophtalmol. 1984;7(10):615-21.
The authors present two cases of neovascular glaucoma following branch retinal vein occlusion. The first case presented bilateral ischaemic branch retinal vein occlusions. In this case there was bilateral impairment of blood flow in the carotido-cerebrovascular system, more marked on the side where the neovascular glaucoma was present. The second case developed neovascular glaucoma following an ischaemic branch retinal vein occlusion of the right eye and had bilateral carotid stenosis that was more complete on the right: where the neovascular glaucoma occurred. The authors analyse the main pathogenic hypothesis of neovascular glaucoma, and suggest that it could be the result of the concurrent action of posterior ocular alterations (like retinal ischaemia following a branch retinal vein occlusion) and of cerebrovascular insufficiency. This second factor could contribute to global ocular ischaemia, thus, involving the anterior vascular structures of the eye as well.
作者报告了两例视网膜分支静脉阻塞后继发新生血管性青光眼的病例。第一例为双侧缺血性视网膜分支静脉阻塞。在该病例中，颈-脑血管系统的血流出现双侧损害，在发生新生血管性青光眼的一侧更为明显。第二例在右眼缺血性视网膜分支静脉阻塞后发生新生血管性青光眼，双侧颈动脉狭窄，右侧更为严重，而新生血管性青光眼正是发生在右侧。作者分析了新生血管性青光眼的主要致病假说，并认为它可能是眼后部改变（如视网膜分支静脉阻塞后的视网膜缺血）和脑血管供血不足共同作用的结果。第二个因素可能导致整体眼部缺血，进而累及眼部的前部血管结构。
