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In vitro reactivity of natural killer (NK) cells against Cryptococcus neoformans.

作者信息

Murphy J W, McDaniel D O

出版信息

J Immunol. 1982 Apr;128(4):1577-83.

PMID:6120974
Abstract
摘要

相似文献

1
In vitro reactivity of natural killer (NK) cells against Cryptococcus neoformans.自然杀伤(NK)细胞对新型隐球菌的体外反应性。
J Immunol. 1982 Apr;128(4):1577-83.
2
Suppression of in vitro maintenance and interferon-mediated augmentation of natural killer cell activity by adherent peritoneal cells from normal mice.正常小鼠腹腔黏附细胞对体外自然杀伤细胞活性维持及干扰素介导增强作用的抑制
J Immunol. 1983 Apr;130(4):1974-9.
3
Comparison between natural reactivity (NR) against Candida albicans and natural killer (NK) activity against YAC-1 tumour cells.针对白色念珠菌的天然反应性(NR)与针对YAC-1肿瘤细胞的自然杀伤(NK)活性之间的比较。
Int J Tissue React. 1983;5(1):73-84.
4
Natural killer cells in mouse lung: surface phenotype, target preference, and response to local influenza virus infection.小鼠肺中的自然杀伤细胞:表面表型、靶标偏好及对局部流感病毒感染的反应。
J Immunol. 1983 Dec;131(6):2699-704.
5
Cellular mechanism of primary anti-Thy-1 antibody responses in vitro induced by uniquely immunogenic thymocyte antigens.由独特免疫原性胸腺细胞抗原体外诱导的原发性抗Thy-1抗体反应的细胞机制。
J Immunol. 1984 Mar;132(3):1100-5.
6
Evaluation of hamster natural cytotoxic cells and vaccinia-induced cytotoxic cells for Thy-1.2 homologue by using a mouse monoclonal alpha-Thy-1.2 antibody.利用小鼠单克隆α-Thy-1.2抗体评估仓鼠自然细胞毒性细胞和痘苗诱导的细胞毒性细胞中的Thy-1.2同源物。
J Immunol. 1982 Nov;129(5):2239-43.
7
Host reactivity against clonable tumor cells and the total tumor cell population, studied in a syngeneic murine lymphoma system.在同基因小鼠淋巴瘤系统中研究宿主对可克隆肿瘤细胞和整个肿瘤细胞群体的反应性。
Acta Pathol Microbiol Immunol Scand C. 1983 Apr;91(2):101-8.
8
Enhancement of tumor metastasis and suppression of natural killer cell activity by beta-estradiol treatment.β-雌二醇治疗增强肿瘤转移并抑制自然杀伤细胞活性。
J Immunol. 1983 Feb;130(2):974-80.
9
Afferent phase production of TNF-alpha is required for the development of protective T cell immunity to Cryptococcus neoformans.产生肿瘤坏死因子-α的传入相对于新型隐球菌保护性T细胞免疫的发展是必需的。
J Immunol. 1996 Nov 15;157(10):4529-36.
10
Regulation of cell-mediated immunity in cryptococcosis. I. Induction of specific afferent T suppressor cells by cryptococcal antigen.隐球菌病中细胞介导免疫的调节。I. 隐球菌抗原诱导特异性传入性T抑制细胞
J Immunol. 1982 Jan;128(1):276-83.

引用本文的文献

1
Genes Influence the Virulence of Cryptococcus neoformans through Contributions beyond Core Autophagy Functions.基因通过超越核心自噬功能的贡献影响新型隐球菌的毒力。
Infect Immun. 2018 Aug 22;86(9). doi: 10.1128/IAI.00069-18. Print 2018 Sep.
2
NK Cell Recognition of Candida glabrata through Binding of NKp46 and NCR1 to Fungal Ligands Epa1, Epa6, and Epa7.自然杀伤细胞通过NKp46和NCR1与真菌配体Epa1、Epa6和Epa7结合来识别光滑念珠菌。
Cell Host Microbe. 2016 Oct 12;20(4):527-534. doi: 10.1016/j.chom.2016.09.008.
3
Innate immune cell response upon Candida albicans infection.
白色念珠菌感染后的固有免疫细胞反应。
Virulence. 2016 Jul 3;7(5):512-26. doi: 10.1080/21505594.2016.1138201. Epub 2016 Apr 14.
4
Host response to Candida albicans bloodstream infection and sepsis.宿主对白色念珠菌血流感染和脓毒症的反应。
Virulence. 2015;6(4):316-26. doi: 10.4161/21505594.2014.988096. Epub 2015 Mar 18.
5
Natural killer cells and antifungal host response.自然杀伤细胞与抗真菌宿主反应。
Clin Vaccine Immunol. 2013 Apr;20(4):452-8. doi: 10.1128/CVI.00606-12. Epub 2013 Jan 30.
6
Host defence to pulmonary mycosis.宿主对肺部真菌病的防御。
Can J Infect Dis. 1999 Mar;10(2):147-55. doi: 10.1155/1999/139326.
7
CpG oligodeoxynucleotides promote the host protective response against infection with Cryptococcus neoformans through induction of interferon-gamma production by CD4+ T cells.CpG寡脱氧核苷酸通过诱导CD4 + T细胞产生干扰素-γ,促进宿主对新型隐球菌感染的保护性反应。
Clin Exp Immunol. 2005 May;140(2):220-9. doi: 10.1111/j.1365-2249.2005.02772.x.
8
Primary dendritic cells phagocytose Cryptococcus neoformans via mannose receptors and Fcgamma receptor II for presentation to T lymphocytes.初级树突状细胞通过甘露糖受体和Fcγ受体II吞噬新型隐球菌,以呈递给T淋巴细胞。
Infect Immun. 2002 Nov;70(11):5972-81. doi: 10.1128/IAI.70.11.5972-5981.2002.
9
Both CD4+ and CD8+ human lymphocytes are activated and proliferate in response to Cryptococcus neoformans.CD4+ 和 CD8+ 人类淋巴细胞在新型隐球菌的刺激下均被激活并增殖。
Immunology. 1997 Oct;92(2):194-200. doi: 10.1046/j.1365-2567.1997.00345.x.
10
Direct interactions of human natural killer cells with Cryptococcus neoformans inhibit granulocyte-macrophage colony-stimulating factor and tumor necrosis factor alpha production.人类自然杀伤细胞与新型隐球菌的直接相互作用会抑制粒细胞巨噬细胞集落刺激因子和肿瘤坏死因子α的产生。
Infect Immun. 1997 Nov;65(11):4564-71. doi: 10.1128/iai.65.11.4564-4571.1997.