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2
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Adherence of Streptococcus sanguis clinical isolates to smooth surfaces and interactions of the isolates with Streptococcus mutans glucosyltransferase.血链球菌临床分离株对光滑表面的黏附以及这些分离株与变形链球菌葡糖基转移酶的相互作用。
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Complete genome and transcriptomes of Streptococcus parasanguinis FW213: phylogenic relations and potential virulence mechanisms.副血链球菌 FW213 的全基因组和转录组：系统发育关系和潜在的毒力机制。
PLoS One. 2012;7(4):e34769. doi: 10.1371/journal.pone.0034769. Epub 2012 Apr 18.
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本文引用的文献

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Classification of the streptococci of subacute bacterial endocarditis.亚急性细菌性心内膜炎链球菌的分类
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2
Biochemical and morphological aspects of extracellular polysaccharides produced by cariogenic streptococci.致龋性链球菌产生的细胞外多糖的生化及形态学方面
Helv Odontol Acta. 1967 Oct;11(2):131-52.
3
Dextran-induced agglutination of Streptococcus mutans, and its potential role in the formation of microbial dental plaques.右旋糖酐诱导的变形链球菌凝集及其在微生物牙菌斑形成中的潜在作用。
J Bacteriol. 1969 May;98(2):341-6. doi: 10.1128/jb.98.2.341-346.1969.
4
Proportions of Streptococcus sanguis, an organism associated with subacute bacterial endocarditis, in human feces and dental plaque.人类粪便和牙菌斑中与亚急性细菌性心内膜炎相关的血链球菌比例。
Infect Immun. 1971 Nov;4(5):658-9. doi: 10.1128/iai.4.5.658-659.1971.
5
Synthesis of extracellular dextran by cariogenic bacteria and its presence in human dental plaque.致龋菌合成细胞外葡聚糖及其在人类牙菌斑中的存在
Arch Oral Biol. 1967 Jan;12(1):11-23. doi: 10.1016/0003-9969(67)90137-9.
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Infective endocarditis--1973.感染性心内膜炎——1973年。
Prog Cardiovasc Dis. 1973 Nov-Dec;16(3):239-74. doi: 10.1016/s0033-0620(73)80001-5.
7
Mechanism of adherence of Streptococcus mutans to smooth surfaces. I. Roles of insoluble dextran-levan synthetase enzymes and cell wall polysaccharide antigen in plaque formation.变形链球菌对光滑表面的黏附机制。I. 不溶性葡聚糖-果聚糖合成酶和细胞壁多糖抗原在菌斑形成中的作用。
Infect Immun. 1973 Oct;8(4):555-62. doi: 10.1128/iai.8.4.555-562.1973.
8
Regulation of dextransucrase formation by Streptococcus sanguis.血链球菌对葡聚糖蔗糖酶形成的调控
Caries Res. 1973;7(2):89-101. doi: 10.1159/000259834.
9
Adhesion of dextran to Streptococcus mutans.葡聚糖与变形链球菌的黏附作用。
J Gen Microbiol. 1974 Apr;81(2):485-9. doi: 10.1099/00221287-81-2-485.
10
Mechanism of adherence of Streptococcus mutans to smooth surfaces. II. Nature of the binding site and the adsorption of dextran-levan synthetase enzymes on the cell-wall surface of the streptococcus.变形链球菌对光滑表面的黏附机制。II. 结合位点的性质以及葡聚糖-果聚糖合成酶在链球菌细胞壁表面的吸附
Infect Immun. 1974 Feb;9(2):419-29. doi: 10.1128/iai.9.2.419-429.1974.

右旋糖酐分子量对血链球菌黏附于受损心脏瓣膜的影响。

Effects of molecular weight of dextran on the adherence of Streptococcus sanguis to damaged heart valves.

作者信息

Ramirez-Ronda C H

出版信息

Infect Immun. 1980 Jul;29(1):1-7. doi: 10.1128/iai.29.1.1-7.1980.

DOI:10.1128/iai.29.1.1-7.1980
PMID:6156909
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC551065/
Abstract

Dextran-producing streptococci such as Streptococcus sanguis are the organisms most frequently associated with infective endocarditis in humans. A series of experiments was designed to study how the molecular weight of dextrans affects the adherence of an endocarditis strain of S. sanguis to canine heart valves covered with platelets and fibrin. The data indicated that this adherence was dependent on dextrans of high molecular weight, such as dextran T-2000 or glucans isolated from S. sanguis or S. mutans. The adherence properties of the strain studied were not modified by prior exposure of the bacterial cells of valve leaflets to high-molecular-weight dextrans. Preexposure of bacterial cells or valve leaflets to low-molecular-weight dextrans decreased their adherence. Low-molecular-weight dextrans interfered with adherence of dextran-positive strains to damaged heart valves.

摘要

产生葡聚糖的链球菌，如血链球菌，是人类感染性心内膜炎最常涉及的微生物。设计了一系列实验来研究葡聚糖的分子量如何影响血链球菌心内膜炎菌株对覆盖有血小板和纤维蛋白的犬心瓣膜的黏附。数据表明，这种黏附依赖于高分子量的葡聚糖，如葡聚糖T - 2000或从血链球菌或变形链球菌中分离出的葡聚糖。所研究菌株的黏附特性不会因细菌细胞或瓣膜小叶预先暴露于高分子量葡聚糖而改变。细菌细胞或瓣膜小叶预先暴露于低分子量葡聚糖会降低它们的黏附。低分子量葡聚糖会干扰葡聚糖阳性菌株对受损心脏瓣膜的黏附。