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1
Vanadate uptake in Neurospora crassa occurs via phosphate transport system II.粗糙脉孢菌中钒酸盐的摄取是通过磷酸转运系统II进行的。
J Bacteriol. 1983 Jan;153(1):286-91. doi: 10.1128/jb.153.1.286-291.1983.
2
Vanadate-resistant mutants of Neurospora crassa are deficient in a high-affinity phosphate transport system.粗糙脉孢菌的钒酸盐抗性突变体在高亲和力磷酸盐转运系统中存在缺陷。
J Bacteriol. 1983 Jan;153(1):292-6. doi: 10.1128/jb.153.1.292-296.1983.
3
Genetic regulation of phosphate transport system II in Neurospora.粗糙脉孢菌中磷酸盐转运系统II的遗传调控
Biochim Biophys Acta. 1975 Nov 17;413(1):95-103. doi: 10.1016/0005-2736(75)90061-9.
4
Adaptive changes in phosphate uptake by the fungus Neurospora crassa in response to phosphate supply.粗糙脉孢菌对磷供应的响应中磷吸收的适应性变化。
J Bacteriol. 1977 Nov;132(2):520-5. doi: 10.1128/jb.132.2.520-525.1977.
5
Kinetic characterization of the two phosphate uptake systems in the fungus Neurospora crassa.粗糙脉孢菌中两种磷酸盐吸收系统的动力学特性
J Bacteriol. 1977 Nov;132(2):511-9. doi: 10.1128/jb.132.2.511-519.1977.
6
Mechanisms controlling the two phosphate uptake systems in Neurospora crassa.控制粗糙脉孢菌中两种磷酸盐吸收系统的机制。
J Bacteriol. 1979 Jul;139(1):195-204. doi: 10.1128/jb.139.1.195-204.1979.
7
Phosphate transport in Neurospora. Derepression of a high-affinity transport system during phosphorus starvation.粗糙脉孢菌中的磷酸盐转运。磷饥饿期间高亲和力转运系统的去阻遏作用。
Biochim Biophys Acta. 1975 May 21;389(3):541-9. doi: 10.1016/0005-2736(75)90164-9.
8
Phosphate transport in Neurospora. Kinetic characterization of a constitutive, low-affinity transport system.粗糙脉孢菌中的磷酸盐转运。一种组成型低亲和力转运系统的动力学特征。
Biochim Biophys Acta. 1974 Dec 24;373(3):369-82. doi: 10.1016/0005-2736(74)90017-0.
9
The properties of arginine transport in vacuolar membrane vesicles of Neurospora crassa.粗糙脉孢菌液泡膜囊泡中精氨酸转运的特性。
J Biol Chem. 1986 Jul 5;261(19):8877-82.
10
Does cycloheximide-induced loss of phosphate uptake activity in Neurospora crassa reflect rapid turnover?环己酰亚胺诱导的粗糙脉孢菌磷酸盐摄取活性丧失是否反映了快速周转?
J Bacteriol. 1978 Jun;134(3):1176-8. doi: 10.1128/jb.134.3.1176-1178.1978.

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Transcriptome analysis provides new insights into the tolerance and reduction of Lysinibacillus fusiformis 15-4 to hexavalent chromium.转录组分析为溶杆菌 15-4 耐受和还原六价铬提供了新的见解。
Appl Microbiol Biotechnol. 2021 Oct;105(20):7841-7855. doi: 10.1007/s00253-021-11586-3. Epub 2021 Sep 21.
2
Dissection of differential vanadate sensitivity in two Ogataea species links protein glycosylation and phosphate transport regulation.在两个 Ogataea 物种中对钒酸盐敏感性的差异进行剖析,将蛋白质糖基化和磷酸盐转运调控联系起来。
Sci Rep. 2018 Nov 6;8(1):16428. doi: 10.1038/s41598-018-34888-5.
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Expression Changes in Metal-Resistance Genes in Microbacterium liquefaciens Under Nickel and Vanadium Exposure.镍和钒暴露下液化微杆菌中金属抗性基因的表达变化
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Bioaccumulation of Vanadium by Vanadium-Resistant Bacteria Isolated from the Intestine of Ascidia sydneiensis samea.从萨氏海鞘肠道中分离出的抗钒细菌对钒的生物累积作用
Mar Biotechnol (NY). 2016 Jun;18(3):359-71. doi: 10.1007/s10126-016-9697-5. Epub 2016 May 13.
5
Vanadate influence on metabolism of sugar phosphates in fungus Phycomyces blakesleeanus.钒酸盐对毛霉属真菌中磷酸糖代谢的影响。
PLoS One. 2014 Jul 18;9(7):e102849. doi: 10.1371/journal.pone.0102849. eCollection 2014.
6
Analysis of microsomal flavoproteins from Phycomyces sporangiophores: Candidates for the blue-light photoreceptor.从 Phycomyces 孢子梗中分析微粒体黄素蛋白:候选蓝光光受体。
Planta. 1985 Apr;163(4):506-16. doi: 10.1007/BF00392708.
7
Effect of Na(3)VO(4) on the P State of Nitella translucens.焦磷酸钠对鳗草 P 态的影响。
Plant Physiol. 1991 May;96(1):91-7. doi: 10.1104/pp.96.1.91.
8
Vanadate-resistant mutants of Neurospora crassa are deficient in a high-affinity phosphate transport system.粗糙脉孢菌的钒酸盐抗性突变体在高亲和力磷酸盐转运系统中存在缺陷。
J Bacteriol. 1983 Jan;153(1):292-6. doi: 10.1128/jb.153.1.292-296.1983.
9
Kinetics of nutrient-limited transport and microbial growth.营养物质限制运输与微生物生长的动力学
Microbiol Rev. 1985 Sep;49(3):270-97. doi: 10.1128/mr.49.3.270-297.1985.
10
Isolation and characterization of vanadate-resistant mutants of Saccharomyces cerevisiae.酿酒酵母钒酸盐抗性突变体的分离与鉴定
J Bacteriol. 1985 Nov;164(2):611-7. doi: 10.1128/jb.164.2.611-617.1985.

本文引用的文献

1
Vanadate inhibition of auxin-enhanced H secretion and elongation in pea epicotyls and oat coleoptiles.钒酸盐抑制生长素增强的豌豆上胚轴和燕麦胚芽鞘的 H 分泌和伸长。
Proc Natl Acad Sci U S A. 1980 Dec;77(12):7242-6. doi: 10.1073/pnas.77.12.7242.
2
POTASSIUM TRANSPORT IN NEUROSPORA. I. INTRACELLULAR SODIUM AND POTASSIUM CONCENTRATIONS, AND CATION REQUIREMENTS FOR GROWTH.粗糙脉孢菌中的钾转运。I. 细胞内钠和钾浓度以及生长所需的阳离子
Biochim Biophys Acta. 1964 Nov 29;88:578-92.
3
The proton-translocating ATPase of the fungal plasma membrane.真菌质膜的质子转运ATP酶。
Biochim Biophys Acta. 1981 Dec 30;639(3-4):197-223. doi: 10.1016/0304-4173(81)90010-0.
4
Vanadate-resistant mutants of Neurospora crassa are deficient in a high-affinity phosphate transport system.粗糙脉孢菌的钒酸盐抗性突变体在高亲和力磷酸盐转运系统中存在缺陷。
J Bacteriol. 1983 Jan;153(1):292-6. doi: 10.1128/jb.153.1.292-296.1983.
5
Inorganic phosphate transport in Escherichia coli: involvement of two genes which play a role in alkaline phosphatase regulation.大肠杆菌中的无机磷酸盐转运:两个在碱性磷酸酶调节中起作用的基因的参与。
J Bacteriol. 1973 Feb;113(2):529-39. doi: 10.1128/jb.113.2.529-539.1973.
6
Phosphate transport in Neurospora. Kinetic characterization of a constitutive, low-affinity transport system.粗糙脉孢菌中的磷酸盐转运。一种组成型低亲和力转运系统的动力学特征。
Biochim Biophys Acta. 1974 Dec 24;373(3):369-82. doi: 10.1016/0005-2736(74)90017-0.
7
Phosphate transport in Neurospora. Derepression of a high-affinity transport system during phosphorus starvation.粗糙脉孢菌中的磷酸盐转运。磷饥饿期间高亲和力转运系统的去阻遏作用。
Biochim Biophys Acta. 1975 May 21;389(3):541-9. doi: 10.1016/0005-2736(75)90164-9.
8
Implications of some genetic control mechanisms in Neurospora.粗糙脉孢菌中一些遗传控制机制的影响
Microbiol Rev. 1979 Sep;43(3):361-83. doi: 10.1128/mr.43.3.361-383.1979.
9
The fate of cytoplasmic vanadium. Implications on (NA,K)-ATPase inhibition.细胞质钒的命运。对(钠,钾)-ATP酶抑制的影响。
J Biol Chem. 1979 Mar 25;254(6):1781-4.
10
A characterization of vanadate interactions with the (Na,K)-ATPase. Mechanistic and regulatory implications.钒酸盐与(钠,钾)-ATP酶相互作用的特征。机制及调节意义。
J Biol Chem. 1978 Oct 25;253(20):7361-8.

粗糙脉孢菌中钒酸盐的摄取是通过磷酸转运系统II进行的。

Vanadate uptake in Neurospora crassa occurs via phosphate transport system II.

作者信息

Bowman B J

出版信息

J Bacteriol. 1983 Jan;153(1):286-91. doi: 10.1128/jb.153.1.286-291.1983.

DOI:10.1128/jb.153.1.286-291.1983
PMID:6217192
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC217368/
Abstract

Vanadate, a potent inhibitor of plasma membrane ATPases, is taken up by Neurospora crassa only when cells are growing in alkaline medium and starving for phosphate. The appearance of a vanadate uptake system (Km = 8.2 microM; Vmax = 0.15 mmol/min per liter of cell water) occurs under the same conditions required for derepression of a high-affinity phosphate transport system. Phosphate is a competitive inhibitor of vanadate uptake, and vanadate is a competitive inhibitor of phosphate uptake. Furthermore, mutant strains which are either partially constitutive or non-derepressible for the high-affinity phosphate transport system are also partially constitutive or non-derepressible for vanadate uptake. These data indicate that vanadate enters the cell via phosphate transport system II.

摘要

钒酸盐是质膜ATP酶的一种有效抑制剂,只有当细胞在碱性培养基中生长且缺乏磷酸盐时,粗糙脉孢菌才会摄取钒酸盐。钒酸盐摄取系统(Km = 8.2微摩尔;Vmax = 0.15毫摩尔/每分钟每升细胞水)的出现与高亲和力磷酸盐转运系统去阻遏所需的相同条件下发生。磷酸盐是钒酸盐摄取的竞争性抑制剂,而钒酸盐是磷酸盐摄取的竞争性抑制剂。此外,对于高亲和力磷酸盐转运系统部分组成型或不可去阻遏的突变菌株,对钒酸盐摄取也是部分组成型或不可去阻遏的。这些数据表明钒酸盐通过磷酸盐转运系统II进入细胞。