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1
Identification of an I-J+ antigen-presenting cell required for third order suppressor cell activation.鉴定三级抑制细胞激活所需的I-J+抗原呈递细胞。
J Exp Med. 1983 Jan 1;157(1):353-8. doi: 10.1084/jem.157.1.353.
2
Requirements for suppressor T cell activation.抑制性T细胞激活的要求。
J Immunol. 1984 Sep;133(3):1137-41.
3
Mechanism responsible for the induction of I-J restriction on TS3 suppressor cells.TS3抑制细胞上I-J限制诱导的负责机制。
J Exp Med. 1982 Nov 1;156(5):1502-15. doi: 10.1084/jem.156.5.1502.
4
I-J-restricted interactions in the generation of azobenzenearsonate-specific suppressor T cells.偶氮苯胂酸盐特异性抑制性T细胞产生过程中的I-J限制相互作用。
J Exp Med. 1982 Nov 1;156(5):1325-34. doi: 10.1084/jem.156.5.1325.
5
Functional analysis of cloned macrophage hybridomas. V. Induction of suppressor T cell responses.克隆化巨噬细胞杂交瘤的功能分析。V. 抑制性T细胞反应的诱导
J Immunol. 1986 Oct 1;137(7):2145-51.
6
The role of adherent accessory cells in the generation of effector suppressor T cells.黏附辅助细胞在效应性抑制性T细胞产生中的作用。
J Immunol. 1986 Dec 15;137(12):3717-25.
7
Characterization of the accessory cells involved in suppressor T cell induction.参与抑制性T细胞诱导的辅助细胞的特征分析。
J Immunol. 1985 Mar;134(3):1374-80.
8
Suppressor T cells, antigen-presenting cells and the role of I-J restriction in oral tolerance to ovalbumin.抑制性T细胞、抗原呈递细胞以及I-J限制在对卵清蛋白的口服耐受中的作用。
Immunology. 1988 May;64(1):141-5.
9
Requirements for suppressor cell activation. Role of accessory cells.抑制细胞激活的要求。辅助细胞的作用。
J Immunol. 1989 Apr 1;142(7):2192-9.
10
Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. XIII. Characterization of a third T cell population involved in suppression of in vitro PFC responses.对半抗原4-羟基-3-硝基苯乙酰的特异性T细胞应答。十三。参与体外PFC反应抑制的第三种T细胞群体的特征。
J Immunol. 1982 Mar;128(3):1260-6.

引用本文的文献

1
I-J determinants as restriction and activation signals.作为限制和激活信号的I-J决定簇。
Surv Immunol Res. 1983;2(3):233-6. doi: 10.1007/BF02918419.
2
Anti-I-J alloantisera elicited by immunization of B10.A(3R) (I-Jb) mice with bone marrow-derived macrophages from B10.A(5R) (I-Jk) mice.通过用来自B10.A(5R)(I-Jk)小鼠的骨髓来源巨噬细胞免疫B10.A(3R)(I-Jb)小鼠而产生的抗I-J同种抗血清。
Immunology. 1986 Mar;57(3):443-9.
3
Monocyte subsets in the production of inhibitory factor by Candida albicans-activated human T cells.白色念珠菌激活的人T细胞产生抑制因子过程中的单核细胞亚群
Immunology. 1985 Oct;56(2):373-6.
4
Anti-I-A antibody modulation of lymphocyte traffic in hapten-stimulated inbred mice.抗I-A抗体对经半抗原刺激的近交系小鼠淋巴细胞迁移的调节作用
Immunology. 1987 Nov;62(3):471-5.
5
Visualization of anti-I-J antibody binding sites on bone marrow-derived macrophages.
Immunol Res. 1987;6(3):210-4. doi: 10.1007/BF02918092.
6
Immunophenotypic characterization of the cutaneous exanthem of SIV-infected rhesus monkeys. Apposition of degenerative Langerhans cells and cytotoxic lymphocytes during the development of acquired immunodeficiency syndrome.感染猴免疫缺陷病毒(SIV)的恒河猴皮肤疹的免疫表型特征。获得性免疫缺陷综合征发展过程中退行性朗格汉斯细胞与细胞毒性淋巴细胞的并置。
Am J Pathol. 1987 Feb;126(2):199-207.
7
Advances in the immunobiology of the skin. Implications for cutaneous malignancies.皮肤免疫生物学的进展。对皮肤恶性肿瘤的影响。
Cancer Metastasis Rev. 1986;5(2):167-78. doi: 10.1007/BF00046429.
8
I-J-positive cloned macrophages as accessory cells for the induction of suppressor T cells in vitro.I-J阳性克隆巨噬细胞作为体外诱导抑制性T细胞的辅助细胞。
Immunol Res. 1986;5(2):106-16. doi: 10.1007/BF02917585.
9
Accessory cell presentation of hapten-modified self.半抗原修饰自身的辅助细胞呈递
Surv Immunol Res. 1985;4(4):303-12. doi: 10.1007/BF02918738.
10
Mechanism of regulation of immune responses by in vivo administration of monoclonal anti-I-A antibodies.体内给予单克隆抗I-A抗体对免疫反应的调节机制。
Surv Immunol Res. 1985;4(3):173-8. doi: 10.1007/BF02918670.

本文引用的文献

1
Hapten-specific T suppressor factor recognizes both hapten and I-J region products on haptenized spleen cells.半抗原特异性T抑制因子可识别半抗原化脾细胞上的半抗原和I-J区产物。
Nature. 1982 Jun 3;297(5865):411-3. doi: 10.1038/297411a0.
2
Helper and suppressor T cell factors.辅助性和抑制性T细胞因子。
Springer Semin Immunopathol. 1980 May;3(1):93-127. doi: 10.1007/BF00199927.
3
Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. I. Alloreactive suppressor cells rather than killer T cells appear to be the decisive effector cells in lethal graft-vs.-host disease.急性和慢性移植物抗宿主病中的同种抑制性T细胞和同种辅助性T细胞。I. 在致死性移植物抗宿主病中,起决定性作用的效应细胞似乎是同种反应性抑制细胞而非杀伤性T细胞。
J Exp Med. 1982 May 1;155(5):1501-22. doi: 10.1084/jem.155.5.1501.
4
Specialized antigen-presenting cells. Splenic dendritic cells and peritoneal-exudate cells induced by mycobacteria activate effector T cells that are resistant to suppression.特异性抗原呈递细胞。由分枝杆菌诱导产生的脾脏树突状细胞和腹腔渗出细胞可激活对抑制具有抗性的效应T细胞。
J Exp Med. 1982 May 1;155(5):1344-56. doi: 10.1084/jem.155.5.1344.
5
Antigen- and receptor-driven regulatory mechanisms. IV. Idiotype-bearing I-J+ suppressor T cell factors induce second-order suppressor T cells which express anti-idiotypic receptors.抗原和受体驱动的调节机制。IV. 携带独特型的I-J+抑制性T细胞因子诱导表达抗独特型受体的二级抑制性T细胞。
J Exp Med. 1980 May 1;151(5):1183-95. doi: 10.1084/jem.151.5.1183.
6
Antigen- and receptor-driven regulatory mechanisms. III. Induction of delayed type hypersensitivity to azobenzenearsonate with anti-cross-reactive idiotypic antibodies.抗原和受体驱动的调节机制。III. 用抗交叉反应性独特型抗体诱导对偶氮苯砷酸盐的迟发型超敏反应。
J Exp Med. 1980 Apr 1;151(4):896-909. doi: 10.1084/jem.151.4.896.
7
The CBA/N mouse strain: an experimental model illustrating the influence of the X-chromosome on immunity.CBA/N小鼠品系:一个说明X染色体对免疫影响的实验模型。
Adv Immunol. 1982;33:1-71. doi: 10.1016/s0065-2776(08)60834-2.
8
I-J-restricted interactions in the generation of azobenzenearsonate-specific suppressor T cells.偶氮苯胂酸盐特异性抑制性T细胞产生过程中的I-J限制相互作用。
J Exp Med. 1982 Nov 1;156(5):1325-34. doi: 10.1084/jem.156.5.1325.
9
In vitro induction of suppressor T-cells in delayed-type hypersensitivity to BCG and an essential role of I-J positive accessory cells.卡介苗迟发型超敏反应中抑制性T细胞的体外诱导及I-J阳性辅助细胞的重要作用。
Immunol Lett. 1982 Jun;4(6):295-9. doi: 10.1016/0165-2478(82)90055-4.
10
Spontaneous remission and acquired resistance to autoimmune encephalomyelitis (EAE) are associated with suppression of T cell reactivity: suppressed EAE effector T cells recovered as T cell lines.自身免疫性脑脊髓炎(EAE)的自发缓解和获得性抗性与T细胞反应性的抑制相关:被抑制的EAE效应T细胞作为T细胞系得以恢复。
J Immunol. 1982 Mar;128(3):1450-7.

鉴定三级抑制细胞激活所需的I-J+抗原呈递细胞。

Identification of an I-J+ antigen-presenting cell required for third order suppressor cell activation.

作者信息

Lowy A, Tominaga A, Drebin J A, Takaoki M, Benacerraf B, Greene M I

出版信息

J Exp Med. 1983 Jan 1;157(1):353-8. doi: 10.1084/jem.157.1.353.

DOI:10.1084/jem.157.1.353
PMID:6217280
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2186903/
Abstract

We have found that an I-J+ I-A- antigen-presenting cell (APC) is required for Ts3 activation in vivo. Together with the I-J restriction previously reported for Ts3 induction (Takaoki, M., M.-S. Sy, A. Tominaga, A. Lowy, M. Tsurifiji, B. Benacerraf, R. Finberg, and M. I. Greene, 1982, J. Exp. Med., 156:1325), it appears that this I-J+ APC is responsible for I-J restriction in the triggering of Ts3. This restriction may be exerted via a pre-Ts3 associative recognition of antigen and I-J encoded determinants, analogous to the T helper recognition of antigen in the context of I-A and I-E determinants.

摘要

我们发现,体内Ts3激活需要I-J⁺I-A⁻抗原呈递细胞(APC)。结合先前报道的Ts3诱导的I-J限制(高木,M.,M.-S. 西,A. 富永,A. 洛伊,M. 鹤桥,B. 贝纳塞拉夫,R. 芬伯格,和M. I. 格林,1982年,《实验医学杂志》,156:1325),看来这种I-J⁺APC在Ts3触发中负责I-J限制。这种限制可能通过Ts3前体对抗原和I-J编码决定簇的联合识别来施加,类似于T辅助细胞在I-A和I-E决定簇背景下对抗原的识别。