Johnston R E, Faulkner P
J Virol. 1978 Jan;25(1):436-8. doi: 10.1128/JVI.25.1.436-438.1978.
Under hypertonic conditions, the penetration of baby hamster kidney cells by Sindbis virus was inhibited. Virus became cell associated, but remained susceptible to neutralization by specific antiserum and to removal by elution. When cells were returned to isotonicity, inhibition was reversed.
在高渗条件下,辛德毕斯病毒对幼仓鼠肾细胞的穿透受到抑制。病毒与细胞结合,但仍易被特异性抗血清中和并通过洗脱去除。当细胞恢复到等渗状态时,抑制作用逆转。