Acetylcholine-induced channels and transmitter release at human endplates. - Suppr | 超能文献

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Acetylcholine-induced channels and transmitter release at human endplates. - Suppr | 超能文献

Acetylcholine-induced channels and transmitter release at human endplates.

作者信息

Cull-Candy S G, Miledi R, Trautmann A

出版信息

Nature. 1978 Jan 5;271(5640):74-5. doi: 10.1038/271074a0.

DOI:10.1038/271074a0

Abstract

摘要

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Acetylcholine-induced channels and transmitter release at human endplates.

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引用本文的文献

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Denervation changes in normal and myasthenia gravis human muscle fibres during organ culture.器官培养过程中正常及重症肌无力患者肌纤维的去神经变化

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Monoclonal antibodies modify acetylcholine-induced ionic channel properties in cultured chick myoballs.单克隆抗体可改变培养的鸡肌球中乙酰胆碱诱导的离子通道特性。

J Membr Biol. 1983;76(2):123-8. doi: 10.1007/BF02000612.

3

Properties of junctional and extrajunctional acetylcholine-receptor channels in organ cultured human muscle fibres.

器官培养的人肌纤维中连接区和连接外乙酰胆碱受体通道的特性

J Physiol. 1982 Dec;333:251-67. doi: 10.1113/jphysiol.1982.sp014452.

4

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J Physiol. 1981 Feb;311:251-66. doi: 10.1113/jphysiol.1981.sp013583.

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A post-natal decrease in acetylcholine channel open time at rat end-plates.大鼠终板处产后乙酰胆碱通道开放时间的减少。

J Physiol. 1980 Jun;303:125-37. doi: 10.1113/jphysiol.1980.sp013275.

6

The acetylcholine channel open time in chick muscle is not decreased following innervation.鸡肌肉中乙酰胆碱通道的开放时间在神经支配后并未缩短。

J Physiol. 1980 Jun;303:111-24. doi: 10.1113/jphysiol.1980.sp013274.

7

On the release of transmitter at normal, myasthenia gravis and myasthenic syndrome affected human end-plates.在正常、重症肌无力及肌无力综合征患者的终板处递质释放情况。

J Physiol. 1980 Feb;299:621-38. doi: 10.1113/jphysiol.1980.sp013145.

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