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黄斑囊样水肿的发病机制:玻璃体视网膜粘连的解剖学考量

Pathogenesis of cystoid macular edema: an anatomic consideration of vitreoretinal adhesions.

作者信息

Sebag J, Balazs E A

出版信息

Surv Ophthalmol. 1984 May;28 Suppl:493-8. doi: 10.1016/0039-6257(84)90231-5.

DOI:10.1016/0039-6257(84)90231-5
PMID:6379948
Abstract

Although it had been widely accepted that vitreous traction plays a key role in the pathogenesis of aphakic cystoid macular edema, vitreoretinal traction is not observable in many patients and other hypotheses have also been investigated. The authors review the present knowledge of the anatomy of the vitreoretinal interface and the pathologic findings in cystoid macular edema. A new hypothesis combining the effects of both traction and inflammation is proposed.

摘要

尽管玻璃体内牵引在无晶状体性黄斑囊样水肿的发病机制中起关键作用已被广泛接受,但在许多患者中并未观察到玻璃体视网膜牵引,并且也对其他假说进行了研究。作者回顾了目前关于玻璃体视网膜界面解剖学以及黄斑囊样水肿病理结果的知识。提出了一种结合牵引和炎症作用的新假说。

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