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C58小鼠中与年龄相关的Lyt-1,2细胞损失导致对乳酸脱氢酶病毒诱导的脑脊髓灰质炎易感性增加。

Age-related loss of Lyt-1,2 cells in C58 mice results in susceptibility to lactic dehydrogenase virus-induced polioencephalomyelitis.

作者信息

Bentley D M, Watson S R, Morris R E

出版信息

Infect Immun. 1983 Sep;41(3):1389-90. doi: 10.1128/iai.41.3.1389-1390.1983.

DOI:10.1128/iai.41.3.1389-1390.1983
PMID:6604028
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC264655/
Abstract

C58 mice (aged greater than or equal to 5 months) are susceptible to age-dependent polioencephalomyelitis, a paralytic central nervous system disease induced by lactic dehydrogenase virus. Susceptibility results from the loss of protective T cells. Data are presented showing a positive correlation between the age-related loss of Lyt-1,2 cells and the development of susceptibility to neuroparalytic lactic dehydrogenase virus infection.

摘要

C58小鼠（年龄大于或等于5个月）易患年龄依赖性脑脊髓灰质炎，这是一种由乳酸脱氢酶病毒引起的麻痹性中枢神经系统疾病。易感性源于保护性T细胞的丧失。所呈现的数据表明，Lyt-1,2细胞与年龄相关的丧失与对神经麻痹性乳酸脱氢酶病毒感染易感性的发展之间存在正相关。

相似文献

1
Age-related loss of Lyt-1,2 cells in C58 mice results in susceptibility to lactic dehydrogenase virus-induced polioencephalomyelitis.C58小鼠中与年龄相关的Lyt-1,2细胞损失导致对乳酸脱氢酶病毒诱导的脑脊髓灰质炎易感性增加。
Infect Immun. 1983 Sep;41(3):1389-90. doi: 10.1128/iai.41.3.1389-1390.1983.
2
T cell subsets required for protection against age-dependent polioencephalomyelitis of C58 mice.抵抗C58小鼠年龄依赖性脑脊髓灰质炎所需的T细胞亚群。
J Immunol. 1982 Feb;128(2):530-4.
3
Kinetics of replication of lactate dehydrogenase-elevating virus in age-dependent polioencephalomyelitis.乳酸脱氢酶升高病毒在年龄依赖性脑脊髓灰质炎中的复制动力学
Intervirology. 1983;19(1):6-15. doi: 10.1159/000149331.
4
Analysis of the Fv1 alleles involved in the susceptibility of mice to lactate dehydrogenase-elevating virus-induced polioencephalomyelitis.对参与小鼠对乳酸脱氢酶升高病毒诱导的脑脊髓灰质炎易感性的Fv1等位基因的分析。
J Neurovirol. 2000 Feb;6(1):89-93. doi: 10.3109/13550280009006386.
5
Susceptibility of C58 mice to paralytic disease induced by lactate dehydrogenase-elevating virus correlates with increased expression of endogenous retrovirus in motor neurons.C58小鼠对乳酸脱氢酶升高病毒诱导的麻痹性疾病的易感性与运动神经元中内源性逆转录病毒表达增加相关。
Microb Pathog. 1988 Oct;5(4):287-96. doi: 10.1016/0882-4010(88)90101-5.
6
Involvement of CD4+ cells in the protection of C58 mouse against polioencephalomyelitis induced by lactate dehydrogenase-elevating virus.
J Neurovirol. 1997 Oct;3(5):380-4. doi: 10.3109/13550289709030752.
7
Mouse strain-specific central nervous system lesions associated with lactate dehydrogenase-elevating virus infection.与乳酸脱氢酶升高病毒感染相关的小鼠品系特异性中枢神经系统病变。
Lab Invest. 1983 Sep;49(3):334-45.
8
Infection of central nervous system cells by ecotropic murine leukemia virus in C58 and AKR mice and in in utero-infected CE/J mice predisposes mice to paralytic infection by lactate dehydrogenase-elevating virus.嗜亲性鼠白血病病毒在C58和AKR小鼠以及子宫内感染的CE/J小鼠中感染中枢神经系统细胞，使小鼠易受乳酸脱氢酶升高病毒的麻痹性感染。
J Virol. 1995 Jan;69(1):308-19. doi: 10.1128/JVI.69.1.308-319.1995.
9
Replication of lactate dehydrogenase-elevating virus in C58 mice and quantification of antiviral antibodies and of tissue virus levels as a function of development of paralytic disease.乳酸脱氢酶升高病毒在C58小鼠中的复制以及抗病毒抗体和组织病毒水平作为麻痹性疾病发展函数的定量分析。
J Gen Virol. 1986 Jan;67 ( Pt 1):27-37. doi: 10.1099/0022-1317-67-1-27.
10
Identification of lactate dehydrogenase-elevating virus as the etiological agent of genetically restricted, age-dependent polioencephalomyelitis of mice.鉴定乳酸脱氢酶升高病毒为小鼠遗传性受限、年龄依赖性脑脊髓灰质炎的病原体。
Infect Immun. 1980 Mar;27(3):979-87. doi: 10.1128/iai.27.3.979-987.1980.

引用本文的文献

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Poliomyelitis in MuLV-infected ICR-SCID mice after injection of basement membrane matrix contaminated with lactate dehydrogenase-elevating virus.注射了被乳酸脱氢酶升高病毒污染的基底膜基质后，MuLV感染的ICR-SCID小鼠发生的脊髓灰质炎
Comp Med. 2011 Oct;61(5):404-11.
2
Lactate dehydrogenase-elevating virus, equine arteritis virus, and simian hemorrhagic fever virus: a new group of positive-strand RNA viruses.乳酸脱氢酶升高病毒、马动脉炎病毒和猴出血热病毒：一组新的正链RNA病毒。
Adv Virus Res. 1992;41:99-192. doi: 10.1016/s0065-3527(08)60036-6.

本文引用的文献

1
Identification of lactate dehydrogenase-elevating virus as the etiological agent of genetically restricted, age-dependent polioencephalomyelitis of mice.鉴定乳酸脱氢酶升高病毒为小鼠遗传性受限、年龄依赖性脑脊髓灰质炎的病原体。
Infect Immun. 1980 Mar;27(3):979-87. doi: 10.1128/iai.27.3.979-987.1980.
2
Failure to demonstrate a role for line Ib tumor-associated surface antigen in the etiology of age-dependent polioencephalomyelitis.
Mol Immunol. 1982 Aug;19(8):983-9. doi: 10.1016/0161-5890(82)90306-6.
3
T cell subsets required for protection against age-dependent polioencephalomyelitis of C58 mice.抵抗C58小鼠年龄依赖性脑脊髓灰质炎所需的T细胞亚群。
J Immunol. 1982 Feb;128(2):530-4.
4
A single major pathway of T-lymphocyte interactions in antigen-specific immune suppression.抗原特异性免疫抑制中T淋巴细胞相互作用的单一主要途径。
Scand J Immunol. 1981;13(1):1-10. doi: 10.1111/j.1365-3083.1981.tb00104.x.
5
Helper and suppressor T cell factors.辅助性和抑制性T细胞因子。
Springer Semin Immunopathol. 1980 May;3(1):93-127. doi: 10.1007/BF00199927.
6
Co-infection by lactic dehydrogenase virus and C-type retrovirus elicits neurological disease.乳酸脱氢酶病毒和C型逆转录病毒的共同感染引发神经疾病。
Nature. 1980 Jul 24;286(5771):398-400. doi: 10.1038/286398a0.
7
Histopathology in immune polioencephalomyelitis in C58 mice.
Arch Neurol. 1973 Jun;28(6):367-70. doi: 10.1001/archneur.1973.00490240027002.
8
Age dependence of immunologically induced central nervous system disease in C58 mice.
Cancer Res. 1970 Jun;30(6):1612-22.
9
Pathogenetic mechanisms in immune polioencephalomyelitis: quantitative evaluation of protective and pathogenic effects of lymphoid cells.
J Immunol. 1976 May;116(5):1332-6.
10
Pathogenetic mechanisms in immune polioencephalomyelitis: induction of disease in immunosuppressed mice.免疫性脑脊髓灰质炎的发病机制：免疫抑制小鼠疾病的诱导
J Immunol. 1976 Feb;116(2):475-81.