Albert D J, Walsh M L, Ryan J, Siemens Y, White R
Behav Neural Biol. 1983 May;38(1):113-9. doi: 10.1016/s0163-1047(83)90444-2.
Rats were either exposed or not exposed to a mouse in their living cage for a 48-hr period. At the end of this time a bilateral lesion was made in the medial accumbens region or in the medial hypothalamus. When tested 2 days postoperatively, the killing frequency among rats that had been exposed to mice preoperatively was not significantly lower than that of rats that were not preoperatively exposed. The ineffectiveness of preoperative experience in suppressing the mouse killing induced by medial accumbens and medial hypothalamic lesions is similar to that found previously with dorsal-median raphe lesions and olfactory bulb lesions and is in contrast to the ease with which preoperative experience prevents mouse killing induced by septal lesions and serotonergic lesions induced by 5,7-dihydroxytryptamine.
将大鼠置于其生活笼中，使其与一只小鼠接触或不接触，持续48小时。在这段时间结束时，在内侧伏隔核区域或内侧下丘脑制造双侧损伤。术后2天进行测试时，术前接触过小鼠的大鼠的捕杀频率并不显著低于术前未接触过小鼠的大鼠。术前经历在抑制由内侧伏隔核和内侧下丘脑损伤诱导的小鼠捕杀方面无效，这与之前发现的背中缝核损伤和嗅球损伤的情况相似，并且与术前经历预防由隔区损伤和5,7 - 二羟基色胺诱导的5-羟色胺能损伤所诱导的小鼠捕杀的容易程度形成对比。
