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[哈钦森-霍顿巨细胞动脉炎和肢根性多肌痛。III. 当前的发病机制和疾病分类学方面]

[Hutchinson-Horton giant cell arteritis and rhizomelic polymyalgia. III. Current pathogenetic and nosographic aspects].

作者信息

Lovisetto P, Marchi L, Gilardi E, Mairano D, Biarese V

出版信息

Minerva Med. 1984 May 7;75(19):1103-10.

PMID:6728260
Abstract

There is much evidence to suggest that temporal arteritis and rhizomelic polymyalgia are both immunological diseases. The classic results of experimental pathology are discussed, together with the relations between rhizomelic polymyalgia and both virus hepatitis B and the HLA system. From the clinical standpoint, it is now agreed that differences in individual response may lead to either a synovial or an arteritic response in both forms. Their association in what Hamrin has called "polymyalgia arteritica" is also common.

摘要

有大量证据表明颞动脉炎和近端多肌痛均为免疫性疾病。本文讨论了实验病理学的经典结果,以及近端多肌痛与乙型病毒性肝炎和HLA系统之间的关系。从临床角度来看,目前人们一致认为个体反应的差异可能导致这两种疾病出现滑膜或动脉炎反应。它们在哈姆林所称的“动脉炎性多肌痛”中的关联也很常见。

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[Hutchinson-Horton giant cell arteritis and rhizomelic polymyalgia. III. Current pathogenetic and nosographic aspects].[哈钦森-霍顿巨细胞动脉炎和肢根性多肌痛。III. 当前的发病机制和疾病分类学方面]
Minerva Med. 1984 May 7;75(19):1103-10.
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