Ivanova E V, Pospelov M E, Strakhovskaia M G, Fraĭkin G Ia
Mikrobiologiia. 1982;51(5):761-4.
While studying the combined action of different UV wavelengths on Saccharomyces cerevisiae (strains XII and Cl-9), it has been found that preliminary irradiation with UV at 334 nm caused photoprotection of the cells against the lethal action of UV at 254 and 313 nm. Postradiation irradiation of strain XII incapable of photoreactivation with UV at 334 nm increased the lethal action of UV at 254 and 313 nm. The mechanisms of the both effects are based on serotonin synthesis induced by the light at 334 nm, as was shown using p-chlorophenylalanine, a specific inhibitor of serotonin synthesis. In strain CI-9 capable of photoreactivation, the postradiation effect of the light at 334 nm depends on the interaction of two different photobiological reactions induced by it, namely, photoreactivation and induced serotonin synthesis.
在研究不同紫外线波长对酿酒酵母（菌株XII和Cl-9）的联合作用时，发现用334nm紫外线进行预照射可使细胞对254nm和313nm紫外线的致死作用产生光保护。用334nm紫外线对无法进行光复活的XII菌株进行辐射后照射，会增加254nm和313nm紫外线的致死作用。这两种效应的机制都基于334nm光诱导的血清素合成，使用血清素合成的特异性抑制剂对氯苯丙氨酸已证明了这一点。在能够进行光复活的CI-9菌株中，334nm光的辐射后效应取决于由其诱导的两种不同光生物学反应的相互作用，即光复活和诱导的血清素合成。
