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Alveolar macrophage lipoxygenase products of arachidonic acid: isolation and recognition as the predominant constituents of the neutrophil chemotactic activity elaborated by alveolar macrophages.

作者信息

Valone F H, Franklin M, Sun F F, Goetzl E J

出版信息

Cell Immunol. 1980 Sep 1;54(2):390-401. doi: 10.1016/0008-8749(80)90219-1.

DOI:10.1016/0008-8749(80)90219-1
PMID:6774816
Abstract
摘要

相似文献

1
Alveolar macrophage lipoxygenase products of arachidonic acid: isolation and recognition as the predominant constituents of the neutrophil chemotactic activity elaborated by alveolar macrophages.肺泡巨噬细胞花生四烯酸的脂氧合酶产物:分离及被确认为肺泡巨噬细胞产生的中性粒细胞趋化活性的主要成分。
Cell Immunol. 1980 Sep 1;54(2):390-401. doi: 10.1016/0008-8749(80)90219-1.
2
Intracellular retention of the 5-lipoxygenase pathway product, leukotriene B4, by human neutrophils activated with unopsonized zymosan.未调理酵母聚糖激活的人中性粒细胞对5-脂氧合酶途径产物白三烯B4的细胞内潴留
J Immunol. 1985 Apr;134(4):2624-30.
3
Relative contribution of leukotriene B4 to the neutrophil chemotactic activity produced by the resident human alveolar macrophage.白三烯B4对人肺泡巨噬细胞产生的中性粒细胞趋化活性的相对贡献。
J Clin Invest. 1987 Oct;80(4):1114-24. doi: 10.1172/JCI113168.
4
Comparison of arachidonic acid metabolism by pulmonary intravascular and alveolar macrophages exposed to particulate and soluble stimuli.暴露于颗粒性和可溶性刺激物的肺血管巨噬细胞和肺泡巨噬细胞的花生四烯酸代谢比较。
Lab Invest. 1989 Oct;61(4):457-66.
5
Lipoxygenase pathways of macrophages.巨噬细胞的脂氧合酶途径
Fed Proc. 1985 Nov;44(14):2933-6.
6
[Influence of endotoxin on the synthesis of lipoxygenase products formed from arachidonic acid by rat alveolar macrophages].[内毒素对大鼠肺泡巨噬细胞由花生四烯酸形成的脂氧合酶产物合成的影响]
Biomed Biochim Acta. 1990;49(5):375-83.
7
Identification of two neutrophil chemotactic peptides produced by porcine alveolar macrophages.猪肺泡巨噬细胞产生的两种中性粒细胞趋化肽的鉴定。
J Biol Chem. 1991 May 5;266(13):8455-63.
8
Tenidap inhibits 5-lipoxygenase product formation in vitro, but this activity is not observed in three animal models.替硝唑在体外可抑制5-脂氧合酶产物的形成,但在三种动物模型中未观察到这种活性。
Inflamm Res. 1997 May;46(5):168-79. doi: 10.1007/s000110050163.
9
Exogenous leukotriene B4 (LTB4) inhibits human neutrophil generation of LTB4 from endogenous arachidonic acid during opsonized zymosan phagocytosis.外源性白三烯B4(LTB4)在调理酵母聚糖吞噬过程中抑制人中性粒细胞从内源性花生四烯酸生成LTB4。
J Pharmacol Exp Ther. 1998 Oct;287(1):150-6.
10
Mediation of leukocyte components of inflammatory reactions by lipoxygenase products of arachidonic acid.花生四烯酸的脂氧合酶产物对炎症反应中白细胞成分的介导作用。
Adv Prostaglandin Thromboxane Leukot Res. 1982;9:273-82.

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The Plasma Oxylipin Signature Provides a Deep Phenotyping of Metabolic Syndrome Complementary to the Clinical Criteria.血浆氧化脂谱特征为代谢综合征提供了一种补充临床标准的深度表型分析。
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Mechanistic insights into cardiovascular protection for omega-3 fatty acids and their bioactive lipid metabolites.ω-3脂肪酸及其生物活性脂质代谢产物对心血管保护作用的机制研究
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Effects of hemodialysis on plasma oxylipins.
血液透析对血浆氧化脂质的影响。
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4
Advances in Our Understanding of Oxylipins Derived from Dietary PUFAs.我们对源自膳食多不饱和脂肪酸的氧化脂质的认识进展
Adv Nutr. 2015 Sep 15;6(5):513-40. doi: 10.3945/an.114.007732. Print 2015 Sep.
5
Glucocorticoid receptors, in human alveolar macrophages and peripheral blood cells.人类肺泡巨噬细胞和外周血细胞中的糖皮质激素受体。
Clin Exp Immunol. 1982 Feb;47(2):505-11.
6
Pulmonary response of fifth component of complement-sufficient and -deficient mice to hyperoxia.补体充足和缺陷小鼠的第五补体成分对高氧的肺反应。
J Clin Invest. 1984 Sep;74(3):956-65. doi: 10.1172/JCI111515.
7
Subcellular localization of the PGE2 synthesis activity in mouse resident peritoneal macrophages.小鼠常驻腹膜巨噬细胞中前列腺素E2合成活性的亚细胞定位
J Exp Med. 1984 Jan 1;159(1):89-102. doi: 10.1084/jem.159.1.89.
8
Enhanced superoxide anion release from phagocytes by muramyl dipeptide or lipopolysaccharide.胞壁酰二肽或脂多糖增强吞噬细胞中超氧阴离子的释放。
Infect Immun. 1983 Feb;39(2):559-64. doi: 10.1128/iai.39.2.559-564.1983.
9
Lipoxygenation of arachidonic acid as a source of polymorphonuclear leukocyte chemotactic factors in synovial fluid and tissue in rheumatoid arthritis and spondyloarthritis.类风湿关节炎和脊柱关节炎中,花生四烯酸的脂氧化作为滑膜液和组织中多形核白细胞趋化因子的来源。
J Clin Invest. 1980 Nov;66(5):1166-70. doi: 10.1172/JCI109947.
10
Metabolites of arachidonic acid.花生四烯酸的代谢产物
Clin Rev Allergy. 1983 Sep;1(3):369-84. doi: 10.1007/BF02991227.