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The effect of extracellular calcium concentration and Ca-antagonist drugs on enzyme release and lactate production by anoxic heart cell cultures.

作者信息

Higgins T J, Allsopp D, Bailey P J

出版信息

J Mol Cell Cardiol. 1980 Sep;12(9):909-27. doi: 10.1016/0022-2828(80)90059-0.

DOI:10.1016/0022-2828(80)90059-0
PMID:6777502
Abstract
摘要

相似文献

1
The effect of extracellular calcium concentration and Ca-antagonist drugs on enzyme release and lactate production by anoxic heart cell cultures.细胞外钙浓度和钙拮抗剂药物对缺氧心脏细胞培养物中酶释放和乳酸生成的影响。
J Mol Cell Cardiol. 1980 Sep;12(9):909-27. doi: 10.1016/0022-2828(80)90059-0.
2
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Cultured neonate rat myocytes as a model for the study of myocardial ischaemic necrosis.培养新生大鼠心肌细胞作为研究心肌缺血坏死的模型。
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Protection of infarcting myocardium by slow channel inhibitors. Comparative effects of verapamil, nifedipine, and diltiazem in the coronary-ligated, isolated working rat heart.慢通道抑制剂对梗死心肌的保护作用。维拉帕米、硝苯地平及地尔硫䓬对冠状动脉结扎的离体工作大鼠心脏的比较效应。
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Effect of extracellular ions and modulators of calcium transport on survival of tert-butyl hydroperoxide exposed cardiac myocytes.细胞外离子及钙转运调节剂对叔丁基过氧化氢处理的心肌细胞存活的影响。
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[The calcium-antagonistic fundamental effects of Verapamil on myocardial fibers and smooth muscle cells of the blood vessels].[维拉帕米对心肌纤维和血管平滑肌细胞的钙拮抗基本作用]
Minerva Med. 1975 May 23;66(38):1827-37.
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Effect of glucose on enzyme release from, and recovery of, the anoxic myocardium.
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10
[Fundamental cardiac and vascular effects of Ca++-antagonistic coronary drugs].
Med Klin. 1975 Oct 17;70(42):1665-74.

引用本文的文献

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Maternal obesity impairs fetal cardiomyocyte contractile function in sheep.母亲肥胖会损害绵羊胎儿心肌细胞的收缩功能。
FASEB J. 2019 Feb;33(2):2587-2598. doi: 10.1096/fj.201800988R. Epub 2018 Oct 5.
2
Reversible and irreversible damage in reoxygenated 'ischemic' ventricular myocytes in culture.培养的再灌注“缺血”心室肌细胞中的可逆性和不可逆性损伤
Mol Cell Biochem. 1996 Jul-Aug;160-161:137-41. doi: 10.1007/BF00240043.
3
Cell injury of cultured rat myocardial cells after reoxygenation of hypoxic cultures in the presence and absence of calcium.
In Vitro. 1984 Aug;20(8):642-6. doi: 10.1007/BF02619614.
4
Studies on oxygen and volume restrictions in cultured cardiac cells. I. A model for ischemia and anoxia with a new approach.培养心肌细胞中氧和容量限制的研究。I. 一种采用新方法的缺血和缺氧模型。
In Vitro Cell Dev Biol. 1985 Sep;21(9):521-5. doi: 10.1007/BF02620845.
5
Effect of verapamil and diltiazem on calcium-dependent electrical activity in cardiac Purkinje fibres.维拉帕米和地尔硫䓬对心脏浦肯野纤维中钙依赖性电活动的影响。
Br J Pharmacol. 1985 May;85(1):89-96. doi: 10.1111/j.1476-5381.1985.tb08834.x.
6
Release of arachidonate from membrane phospholipids in cultured neonatal rat myocardial cells during adenosine triphosphate depletion. Correlation with the progression of cell injury.三磷酸腺苷耗竭期间培养的新生大鼠心肌细胞膜磷脂中花生四烯酸的释放。与细胞损伤进展的相关性。
J Clin Invest. 1985 Jun;75(6):1770-80. doi: 10.1172/JCI111889.
7
Studies on oxygen and volume restriction in cultured cardiac cell: possible rearrangement of sarcolemmal lipid moieties during anoxia and ischemia-like states.培养心肌细胞中氧和容量限制的研究：缺氧和缺血样状态下肌膜脂质部分可能的重排。
Mol Cell Biochem. 1988 Jan;79(1):39-46. doi: 10.1007/BF00229396.
8
Inhibition of the release of arachidonic acid prevents the development of sarcolemmal membrane defects in cultured rat myocardial cells during adenosine triphosphate depletion.抑制花生四烯酸的释放可防止培养的大鼠心肌细胞在三磷酸腺苷耗竭期间肌膜膜缺陷的发展。
J Clin Invest. 1988 Oct;82(4):1333-8. doi: 10.1172/JCI113735.
9
Suppression of snake-venom cardiotoxin-induced cardiomyocyte degeneration by blockage of Ca2+ influx or inhibition of non-lysosomal proteinases.通过阻断Ca2+内流或抑制非溶酶体蛋白酶来抑制蛇毒心脏毒素诱导的心肌细胞变性。
Biochem J. 1988 Nov 15;256(1):89-95. doi: 10.1042/bj2560089.