Sahlin K, Freyschuss U, Hultman E, Sjöholm H
Clin Physiol. 1981 Oct;1(5):495-502. doi: 10.1111/j.1475-097x.1981.tb00916.x.
Subjects inhaled air with 8% CO2 for 10 min. Carbon dioxide tension in arterial blood increased rapidly from about 5 kPa to 7 kPa, whereas venous CO2 tension increased more slowly, reaching the arterial value at the end of the CO2-breathing period. Muscle content of total CO2 (HCO-3, H2CO3, solubilized CO2) did not change during CO2-breathing or the following 15 min of recovery. However, when CO2-breathing was combined with light bicycle exercise, total CO2 increased by 30%. This could be an effect of both increased local circulation and increased endogenous CO2 production in the muscle. It is concluded that 10 min CO2-breathing alone is insufficient to affect CO2 content and intracellular pH in resting skeletal muscle.
受试者吸入含8%二氧化碳的空气10分钟。动脉血中的二氧化碳分压迅速从约5千帕升至7千帕，而静脉血二氧化碳分压上升较慢，在二氧化碳呼吸期结束时达到动脉血的值。在二氧化碳呼吸期间及随后15分钟的恢复过程中，肌肉中总二氧化碳（碳酸氢根、碳酸、溶解的二氧化碳）含量没有变化。然而，当二氧化碳呼吸与轻度自行车运动相结合时，总二氧化碳增加了30%。这可能是局部循环增加和肌肉内源性二氧化碳产生增加共同作用的结果。得出的结论是，仅10分钟的二氧化碳呼吸不足以影响静息骨骼肌中的二氧化碳含量和细胞内pH值。
