Simpson R J, Podolak R, Mangano C A, Foster J R, Dalldorf F G
JAMA. 1983 Jan 21;249(3):390-3.
The mechanism for syncope during pulmonary embolism is not well understood. We describe two patients with transient sinus bradycardia and atrioventricular (AV) block during syncope from recurrent pulmonary embolism. Consciousness was regained each time the rhythm returned to normal. We believe that the syncope and bradyarrhythmia was caused by a parasympathetic reflex, since simultaneous slowing of the sinus rate with concomitant AV block is a common manifestation of increased vagal tone. Such a reflex is consistent with known cardiac reflexes, may occur frequently, and may be one of the mechanisms for syncope in patients with pulmonary embolism.
肺栓塞期间晕厥的机制尚未完全明确。我们描述了两名复发性肺栓塞导致晕厥期间出现短暂性窦性心动过缓和房室传导阻滞的患者。每次心律恢复正常时意识都会恢复。我们认为晕厥和缓慢性心律失常是由副交感神经反射引起的,因为窦性心率同时减慢并伴有房室传导阻滞是迷走神经张力增加的常见表现。这种反射与已知的心脏反射一致,可能经常发生,并且可能是肺栓塞患者晕厥的机制之一。