由弗瑞德白血病病毒体外转化的分化诱导早幼粒细胞白血病细胞系中脾脏病灶形成病毒（SFFV）的释放 - Suppr

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超能文献

Release of spleen focus-forming virus (SFFV) from differentiation inducible promyelocytic leukemia cell lines transformed in vitro by Friend leukemia virus.

作者信息

Greenberger J S, Eckner R J, Ostertag W, Colletta G, Boschetti S, Nagasawa H, Karpas A, Weichselbaum R R, Moloney W C

出版信息

Virology. 1980 Sep;105(2):425-35. doi: 10.1016/0042-6822(80)90043-4.

DOI:10.1016/0042-6822(80)90043-4

PMID:6932782

Abstract

摘要

相似文献

Release of spleen focus-forming virus (SFFV) from differentiation inducible promyelocytic leukemia cell lines transformed in vitro by Friend leukemia virus.由弗瑞德白血病病毒体外转化的分化诱导早幼粒细胞白血病细胞系中脾脏病灶形成病毒（SFFV）的释放

Virology. 1980 Sep;105(2):425-35. doi: 10.1016/0042-6822(80)90043-4.

Virus and cell requirements for Friend virus granulocytic leukemogenesis in long-term bone marrow cultures of NIH swiss [N:NIH(S)] mice.在NIH瑞士[N:NIH(S)]小鼠长期骨髓培养中，Friend病毒诱导粒细胞白血病发生的病毒和细胞需求。

J Natl Cancer Inst. 1980 Apr;64(4):867-78.

Inducibility of spleen focus-forming virus by BrdUrd is controlled by the differentiated state of the cell.5-溴脱氧尿苷对脾集落形成病毒的诱导作用受细胞分化状态的控制。

Proc Natl Acad Sci U S A. 1981 May;78(5):2995-9. doi: 10.1073/pnas.78.5.2995.

Friend erythroleukemia virus complex: role of viral components in modifying erythroid differentiation in mice.Friend红白血病病毒复合体：病毒成分在调节小鼠红系分化中的作用

J Natl Cancer Inst. 1982 Mar;68(3):457-60.

Lack of retrovirus gene expression in somatic cell hybrids of friend cells and teratocarcinoma cells with a teratocarcinoma phenotype.在具有畸胎瘤细胞表型的Friend细胞与畸胎瘤细胞的体细胞杂种中逆转录病毒基因表达缺失。

Mol Cell Biol. 1984 May;4(5):923-30. doi: 10.1128/mcb.4.5.923-930.1984.

Anemia- and polycythemia-inducing isolates of Friend spleen focus-forming virus. Biological and molecular evidence for two distinct viral genomes.弗氏脾脏灶形成病毒的贫血和红细胞增多诱导分离株。两种不同病毒基因组的生物学和分子证据。

J Exp Med. 1980 Jun 1;151(6):1477-92. doi: 10.1084/jem.151.6.1477.

Helper virus is not required for in vitro erythroid transformation of hematopoietic cells by Friend virus.弗氏病毒对造血细胞进行体外红系转化时无需辅助病毒。

Proc Natl Acad Sci U S A. 1980 Sep;77(9):5287-91. doi: 10.1073/pnas.77.9.5287.

Transformation of DBA/2 mouse fetal liver cells infected in vitro by the anemic strain of Friend leukemia virus.体外受贫血型Friend白血病病毒感染的DBA/2小鼠胎儿肝细胞的转化

Proc Natl Acad Sci U S A. 1979 Feb;76(2):962-6. doi: 10.1073/pnas.76.2.962.

Induction of erythroid tumorigenic colonies by Friend helper virus F-MuLV alone and isolation of a new class of friend erythroleukemic cells.单独由弗瑞德辅助病毒F-MuLV诱导红系致瘤集落并分离出一类新的弗瑞德红白血病细胞。

J Cell Physiol Suppl. 1982;1:185-94. doi: 10.1002/jcp.1041130426.

Expression of a transformation-related protein (p53) in the malignant stage of Friend virus-induced diseases.转化相关蛋白（p53）在弗瑞德病毒诱导疾病恶性阶段的表达

J Virol. 1983 Jun;46(3):1022-6. doi: 10.1128/JVI.46.3.1022-1026.1983.

引用本文的文献

Reduced Competitive Repopulation Capacity of Multipotential Hematopoietic Stem Cells in the Bone Marrow of Friend Virus-infected Fv2-resistant Mice.感染Friend病毒的Fv2抗性小鼠骨髓中多能造血干细胞的竞争性再增殖能力降低

In Vivo. 2017 May-Jun;31(3):313-320. doi: 10.21873/invivo.11061.

Hematopoietic cell survival signals are elicited through non-tyrosine-containing sequences in the membrane-proximal region of the erythropoietin receptor (EPOR) by a Stat5-dependent pathway.造血细胞存活信号通过促红细胞生成素受体（EPOR）膜近端区域中不含酪氨酸的序列，由一条依赖Stat5的途径引发。

Exp Hematol. 2003 Dec;31(12):1310-6. doi: 10.1016/j.exphem.2003.08.009.

Cytokine signaling through Stat3 activates integrins, promotes adhesion, and induces growth arrest in the myeloid cell line 32D.细胞因子通过Stat3发出的信号激活整合素，促进黏附，并诱导髓系细胞系32D生长停滞。

J Biol Chem. 2000 Aug 25;275(34):26566-75. doi: 10.1074/jbc.M003495200.

Sequential mutations in the interleukin-3 (IL3)/granulocyte-macrophage colony-stimulating factor/IL5 receptor beta-subunit genes are necessary for the complete conversion to growth autonomy mediated by a truncated beta C subunit.白细胞介素-3（IL3）/粒细胞-巨噬细胞集落刺激因子/IL5受体β亚基基因中的序列突变对于完全转化为由截短的βC亚基介导的生长自主性是必要的。

Mol Cell Biol. 1995 May;15(5):2402-12. doi: 10.1128/MCB.15.5.2402.

Friend disease in vitro.体外Friend病

J Exp Med. 1981 Sep 1;154(3):594-608. doi: 10.1084/jem.154.3.594.

Emergence of tumorigenic cells during the course of Friend virus leukemias.在弗氏病毒白血病病程中致瘤细胞的出现。

Proc Natl Acad Sci U S A. 1981 Jun;78(6):3614-8. doi: 10.1073/pnas.78.6.3614.

Multistep virus-induced leukemogenesis in vitro: description of a model specifying three steps within the myeloblastic malignant process.体外多步骤病毒诱导的白血病发生：一种确定髓母细胞恶性过程中三个步骤的模型描述。

Mol Cell Biol. 1984 Jan;4(1):216-20. doi: 10.1128/mcb.4.1.216-220.1984.

Demonstration of permanent factor-dependent multipotential (erythroid/neutrophil/basophil) hematopoietic progenitor cell lines.永久性因子依赖性多能（红系/中性粒细胞/嗜碱性粒细胞）造血祖细胞系的证明。

Proc Natl Acad Sci U S A. 1983 May;80(10):2931-5. doi: 10.1073/pnas.80.10.2931.

Studies on the in vivo production of a lymphokine activity, interleukin 3 (IL-3) elaborated by lymphocytes and a myeloid leukaemic line in vitro and the fate of IL-3 dependent cell lines.关于淋巴细胞和髓系白血病细胞系在体外产生的一种淋巴因子活性——白细胞介素3（IL-3）的体内生成情况以及IL-3依赖细胞系命运的研究。

Br J Cancer. 1983 Aug;48(2):247-59. doi: 10.1038/bjc.1983.180.

Hemopoietic stem cells and target cells in murine-virus-induced leukemias.小鼠病毒诱导白血病中的造血干细胞和靶细胞。

J Cancer Res Clin Oncol. 1982;104(1-2):1-12. doi: 10.1007/BF00402048.

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