Tabacova S, Balabaeva L
Arch Toxicol Suppl. 1980;4:252-4. doi: 10.1007/978-3-642-67729-8_51.
Exposure by inhalation of pregnant albino rats to 10 and 0.03 mg/m3 carbon disulphide (CS2) does not produce congenital malformations or functional biochemical changes in the neonate, but it affects the postnatal development, causing at 10 mg/m3 impairment of viability, retardation of morphological and sensory development and behavioral deviations (the latter at both exposure levels). These changes are more pronounced in the early postnatal life.
怀孕的白化大鼠吸入10毫克/立方米和0.03毫克/立方米的二硫化碳(CS2)后,未出现先天性畸形或新生儿功能生化变化,但会影响产后发育,在10毫克/立方米时导致生存能力受损、形态和感官发育迟缓以及行为偏差(两种暴露水平下均出现后者)。这些变化在出生后早期更为明显。