Krüger D H, Gola G, Weisshuhn I, Hansen S
J Gen Virol. 1978 Oct;41(1):189-92. doi: 10.1099/0022-1317-41-1-189.
On pre-infection of the host Escherichia coli B with u.v.-inactivated T3 or T7 phage able to express their early genes (like 0.3), B-specific modification of superinfecting, successfully multiplying viruses does not take place. The ocr gene function (gene 0.3) of T3 and T7 not only prevents host-specific DNA restriction but also modification, probably by inhibiting the same late step in the interaction between the restriction enzyme and DNA.
用能够表达其早期基因（如0.3）的经紫外线灭活的T3或T7噬菌体对宿主大肠杆菌B进行预感染时，超感染且能成功增殖的病毒不会发生B特异性修饰。T3和T7的ocr基因功能（基因0.3）不仅可防止宿主特异性DNA限制，还可能通过抑制限制酶与DNA相互作用的同一后期步骤来防止修饰。
