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The role of antibody and complement in lysing virus-infected cells.

作者信息

Sissons J G, Schreiber R D, Cooper N R, Oldstone M B

出版信息

Med Microbiol Immunol. 1982;170(4):221-7. doi: 10.1007/BF02123312.

DOI:10.1007/BF02123312
PMID:7048046
Abstract
摘要

相似文献

1
The role of antibody and complement in lysing virus-infected cells.抗体和补体在裂解病毒感染细胞中的作用。
Med Microbiol Immunol. 1982;170(4):221-7. doi: 10.1007/BF02123312.
2
The complement system: its importance in the host response to viral infection.补体系统：其在宿主对病毒感染的应答中的重要性。
Microbiol Rev. 1982 Mar;46(1):71-85. doi: 10.1128/mr.46.1.71-85.1982.
3
Separate pathways of C activation by measles virus cytotoxic antibodies: subclass analysis and capacity of F(ab) molecules to activate C via the alternative pathway.麻疹病毒细胞毒性抗体激活补体的不同途径：亚类分析及F(ab)分子通过替代途径激活补体的能力
J Immunol. 1978 Sep;121(3):1206-12.
4
Killing of virus-infected cells: the role of antiviral antibody and complement in limiting virus infection.病毒感染细胞的杀伤：抗病毒抗体和补体在限制病毒感染中的作用。
J Infect Dis. 1980 Sep;142(3):442-8. doi: 10.1093/infdis/142.3.442.
5
Lysis of measles virus-infected cells by the purified cytolytic alternative complement pathway and antibody.纯化的溶细胞性替代补体途径和抗体对麻疹病毒感染细胞的裂解作用
J Exp Med. 1979 Sep 19;150(3):445-54. doi: 10.1084/jem.150.3.445.
6
Alternative complement pathway-mediated lysis of measles virus infected cells: induction by IgG antibody bound to individual viral glycoproteins and comparative efficacy of F(ab')2 and Fab' fragments.替代补体途径介导的麻疹病毒感染细胞的裂解：与单个病毒糖蛋白结合的IgG抗体的诱导作用以及F(ab')2和Fab'片段的比较功效
J Immunol. 1979 Nov;123(5):2144-9.
7
[Studies on the interaction between cells infected with measles virus and anti-measles antibody. I. Cytotoxic antibody activity of sera obtained from patients with natural measles and subacute sclerosing panencephalitis by use of chronically infected cell lines].[麻疹病毒感染细胞与抗麻疹抗体相互作用的研究。I. 利用长期感染细胞系检测自然麻疹患者和亚急性硬化性全脑炎患者血清的细胞毒性抗体活性]
Hokkaido Igaku Zasshi. 1986 Jan;61(1):5-14.
8
Escape from immune surveillance during persistent virus infection.持续性病毒感染期间对免疫监视的逃逸。
Adv Exp Med Biol. 1983;162:449-61. doi: 10.1007/978-1-4684-4481-0_41.
9
Immunologic injury of cultured cells infected with measles virus. I. role of IfG antibody and the alternative complement pathway.感染麻疹病毒的培养细胞的免疫损伤。I. 干扰素γ抗体和替代补体途径的作用。
J Exp Med. 1975 Apr 1;141(4):761-74.
10
Antibody-independent activation of the alternative complement pathway by measles virus-infected cells.麻疹病毒感染细胞对替代补体途径的抗体非依赖性激活。
Proc Natl Acad Sci U S A. 1980 Jan;77(1):559-62. doi: 10.1073/pnas.77.1.559.

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Pathogenicity of neutralization escape mutants of mouse hepatitis virus: correlation with T- and B-cell depletions.小鼠肝炎病毒中和逃逸突变体的致病性：与T细胞和B细胞耗竭的相关性。
Res Immunol. 1994 Sep;145(7):553-65. doi: 10.1016/s0923-2494(94)80073-1.

本文引用的文献

1
Antibody-independent activation of the alternative complement pathway by measles virus-infected cells.麻疹病毒感染细胞对替代补体途径的抗体非依赖性激活。
Proc Natl Acad Sci U S A. 1980 Jan;77(1):559-62. doi: 10.1073/pnas.77.1.559.
2
Raji cell injury and subsequent lysis by the purified cytolytic alternative pathway of human complement.人补体的纯化溶细胞替代途径对拉吉细胞的损伤及随后的裂解。
Clin Immunol Immunopathol. 1980 Mar;15(3):384-96. doi: 10.1016/0090-1229(80)90050-1.
3
Killing of virus-infected cells: the role of antiviral antibody and complement in limiting virus infection.
病毒感染细胞的杀伤：抗病毒抗体和补体在限制病毒感染中的作用。
J Infect Dis. 1980 Sep;142(3):442-8. doi: 10.1093/infdis/142.3.442.
4
Antibody-mediated destruction of virus-infected cells.抗体介导的病毒感染细胞的破坏。
Adv Immunol. 1980;29:209-60. doi: 10.1016/s0065-2776(08)60045-0.
5
Killing of virus-infected cells by cytotoxic lymphocytes.细胞毒性淋巴细胞对病毒感染细胞的杀伤作用。
J Infect Dis. 1980 Jul;142(1):114-9. doi: 10.1093/infdis/142.1.114.
6
Lysis of measles virus-infected cells by the purified cytolytic alternative complement pathway and antibody.纯化的溶细胞性替代补体途径和抗体对麻疹病毒感染细胞的裂解作用
J Exp Med. 1979 Sep 19;150(3):445-54. doi: 10.1084/jem.150.3.445.
7
Initiation of the alternative pathway of complement: recognition of activators by bound C3b and assembly of the entire pathway from six isolated proteins.补体替代途径的激活：结合的C3b对激活物的识别以及由六种分离的蛋白质组装整个途径。
Proc Natl Acad Sci U S A. 1978 Aug;75(8):3948-52. doi: 10.1073/pnas.75.8.3948.
8
Regulation by membrane sialic acid of beta1H-dependent decay-dissociation of amplification C3 convertase of the alternative complement pathway.膜唾液酸对替代补体途径中C3转化酶β1H依赖性衰变解离的调节作用。
Proc Natl Acad Sci U S A. 1978 Apr;75(4):1971-5. doi: 10.1073/pnas.75.4.1971.
9
Activation of the alternative complement pathway by human B cell lymphoma lines is associated with Epstein-Barr virus transformation of the cells.人B细胞淋巴瘤系对替代补体途径的激活与细胞的爱泼斯坦-巴尔病毒转化相关。
Eur J Immunol. 1978 Jul;8(7):453-8. doi: 10.1002/eji.1830080702.
10
Mechanism of injury of virus-infected cells by antiviral antibody and complement: participation of IgG, F(ab')2, and the alternative complement pathway.抗病毒抗体和补体对病毒感染细胞的损伤机制：IgG、F(ab')2 及替代补体途径的参与
J Exp Med. 1976 May 1;143(5):1027-41. doi: 10.1084/jem.143.5.1027.