Martin R B, Burr D B
J Biomech. 1982;15(3):137-9. doi: 10.1016/s0021-9290(82)80001-8.
This paper presents a theory describing mechanisms by which repetitive stress initiates remodelling in compact bone. The theory is based on the observation that the lamellar structure of osteons arrests and traps microcracks produced by cyclic loading. Debonding of an osteon by a crack may produce changes in the Haversian canal wall adjacent to the crack which initiate a new secondary osteon. The repair of damaged areas by secondary osteons prevents the accumulation of microdamage due to repetitive loading and protects compact bone from fatigue failure.
本文提出了一种理论，描述重复性应力引发密质骨重塑的机制。该理论基于这样的观察：骨单位的板层结构会阻止并捕获循环载荷产生的微裂纹。裂纹使骨单位脱粘可能会在裂纹附近的哈弗斯管壁产生变化，从而引发新的次级骨单位。次级骨单位对受损区域的修复可防止因重复性载荷导致的微损伤积累，并保护密质骨免于疲劳失效。
